Table 2.

Treatment success in genetic subgroups

Subgroups of PP populationTreatment success: DisC, % (n/n)Treatment success: RIST, % (n/n)Difference (%) (95% CI)P value 
All 84.1 (116/138) 81.3 (109/134) 2.7 (−6.3 to 11.8) .047 
ELN 2022     
Favorable 95.5 (21/22) 100.0 (19/19) −4.5 (−13.7 to 4.6) .46 
Intermediate 81.6 (40/49) 83.3 (60/72) −1.7 (−15.8 to 12.4) .32 
Adverse 82.1 (55/67) 69.8 (30/43) 12.3 (−3.6 to 28.2) .016 
Genetic abnormality     
Mutated NPM1 95.5 (21/22) 79.2 (19/24) 16.3 (−4.2 to 36.8) .021 
Mutated NPM1 and FLT3 wild type 92.9 (13/14) 83.3 (10/12) 9.5 (−15.8 to 34.9) .13 
FLT3-ITD 95.8 (23/24) 66.7 (14/21) 29.2 (6.2 to 52.2) .0018 
Secondary-type mutations  90.9 (20/22) 86.4 (19/22) 4.5 (−14.7 to 23.8) .17 
Complex karyotype 73.5 (25/34) 53.3 (8/15) 20.2 (−7.9 to 48.3) .039 
abn(17p) or TP53 mutations 73.9 (17/23) 70.0 (7/10) 3.9 (−28.4 to 36.2) .29 
Subgroups of PP populationTreatment success: DisC, % (n/n)Treatment success: RIST, % (n/n)Difference (%) (95% CI)P value 
All 84.1 (116/138) 81.3 (109/134) 2.7 (−6.3 to 11.8) .047 
ELN 2022     
Favorable 95.5 (21/22) 100.0 (19/19) −4.5 (−13.7 to 4.6) .46 
Intermediate 81.6 (40/49) 83.3 (60/72) −1.7 (−15.8 to 12.4) .32 
Adverse 82.1 (55/67) 69.8 (30/43) 12.3 (−3.6 to 28.2) .016 
Genetic abnormality     
Mutated NPM1 95.5 (21/22) 79.2 (19/24) 16.3 (−4.2 to 36.8) .021 
Mutated NPM1 and FLT3 wild type 92.9 (13/14) 83.3 (10/12) 9.5 (−15.8 to 34.9) .13 
FLT3-ITD 95.8 (23/24) 66.7 (14/21) 29.2 (6.2 to 52.2) .0018 
Secondary-type mutations  90.9 (20/22) 86.4 (19/22) 4.5 (−14.7 to 23.8) .17 
Complex karyotype 73.5 (25/34) 53.3 (8/15) 20.2 (−7.9 to 48.3) .039 
abn(17p) or TP53 mutations 73.9 (17/23) 70.0 (7/10) 3.9 (−28.4 to 36.2) .29 

Treatment success was defined as CR on day 56 after alloHCT. The differences in success rates between DisC and RIST have been calculated with 95% confidence intervals.

P values <.025 indicate noninferiority of DisC vs RIST with respect to a noninferiority margin of 5%.

Secondary-type mutations comprise ASXL1, BCOR, EZH2, RUNX1, SF3B1, SRSF2, STAG2, U2AF1, and/or ZRSR2 mutations.

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