Table 1.

Patient and disease characteristics

CharacteristicPatients from ASPEN study who transitioned to zanubrutinib (N = 47)
At parent study enrollment: BGB-3111-302 (ASPEN)At LTE enrollment: BGB-3111-LTE1 (LTE1)
Age, median (range), y 68 (38-84) 73 (44-89) 
<65, n (%) 16 (34) 8 (17) 
≥65, n (%) 22 (46.8) 21 (44.7) 
≥75, n (%) 9 (19.1) 18 (38.3) 
Male sex, n (%) 34 (72.3) 34 (72.3) 
Pre-BTK inhibitor treatment status, n (%)   
Treatment naive 10 (21.3)  
Relapsed/refractory 37 (78.7)  
Previous lines of therapy, median (range) 1 (1-6)  
ECOG performance status, n (%)   
25 (53) 27 (57.4) 
21 (45) 17 (36.2) 
1 (2.1) 1 (2.1) 
Missing 2 (4.3) 
CXCR4 mutation status, n (%)   
WHIM FS 5 (10.6)  
WHIM NS 2 (4.3)  
WT 38 (80.9)  
Unknown 2 (4.3)  
TP53 mutation, n (%) 8 (17.0)  
CharacteristicPatients from ASPEN study who transitioned to zanubrutinib (N = 47)
At parent study enrollment: BGB-3111-302 (ASPEN)At LTE enrollment: BGB-3111-LTE1 (LTE1)
Age, median (range), y 68 (38-84) 73 (44-89) 
<65, n (%) 16 (34) 8 (17) 
≥65, n (%) 22 (46.8) 21 (44.7) 
≥75, n (%) 9 (19.1) 18 (38.3) 
Male sex, n (%) 34 (72.3) 34 (72.3) 
Pre-BTK inhibitor treatment status, n (%)   
Treatment naive 10 (21.3)  
Relapsed/refractory 37 (78.7)  
Previous lines of therapy, median (range) 1 (1-6)  
ECOG performance status, n (%)   
25 (53) 27 (57.4) 
21 (45) 17 (36.2) 
1 (2.1) 1 (2.1) 
Missing 2 (4.3) 
CXCR4 mutation status, n (%)   
WHIM FS 5 (10.6)  
WHIM NS 2 (4.3)  
WT 38 (80.9)  
Unknown 2 (4.3)  
TP53 mutation, n (%) 8 (17.0)  

ECOG, Eastern Cooperative Oncology Group; FS, frameshift mutation; NS, nonsense mutation; WHIM, warts, hypogammaglobulinemia, infections, myelokathexis; WT, wild type.

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