Table 2.

Clinical characteristics of patients analyzed late after allogeneic SCT

VariablePatients, n = 18
Age, y, median (range) 43.5 (17-58)  
Patient sex  
 Male 
 Female 9  
Underlying disease  
 CML 
 AML 4  
 ALL 4  
 NHL 4  
 MDS 
Patient/donor HCMV-serologic status  
 +/+ 
 −/+ 1  
 +/− 4  
 −/− 4  
Conditioning regimen  
 TBI/CY ± VP16 11  
 BU/CY ± ARA-C 
 TBI/FLU/CY 2  
Type of donor  
 HLA-identical family donor 10  
 Matched unrelated 6  
 Mismatch family 
Acute GVHD  
 Grades 0/I 11  
 Grade II 
 Grades III/IV 3  
Chronic GVHD  
 None/limited disease 12  
 Extensive disease 
VariablePatients, n = 18
Age, y, median (range) 43.5 (17-58)  
Patient sex  
 Male 
 Female 9  
Underlying disease  
 CML 
 AML 4  
 ALL 4  
 NHL 4  
 MDS 
Patient/donor HCMV-serologic status  
 +/+ 
 −/+ 1  
 +/− 4  
 −/− 4  
Conditioning regimen  
 TBI/CY ± VP16 11  
 BU/CY ± ARA-C 
 TBI/FLU/CY 2  
Type of donor  
 HLA-identical family donor 10  
 Matched unrelated 6  
 Mismatch family 
Acute GVHD  
 Grades 0/I 11  
 Grade II 
 Grades III/IV 3  
Chronic GVHD  
 None/limited disease 12  
 Extensive disease 

CML indicates chronic myeloid leukemia; AML, acute myeloid leukemia; ALL, acute lymphoblastic leukemia; NHL, non-Hodgkin lymphoma; MDS, myelodysplastic syndrome; HCMV, human cytomegalovirus; TBI, total body irradiation; CY, cyclophosphamide; VP16, etoposide; BU, busulfan; FLU, fludarabine; and ARA-C, cytosinarabinoside.

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