Impaired XBP1 splicing downstream of SRP54 mutations. During physiological conditions (A), the signal recognition particle 54 (SRP54) facilitates the translocation of the ribosome-associated unspliced XBP1 (XBP1u) to the ER where it is spliced (XBP1s). In pathological conditions (B), SRP54 mutations found in patients act in a dominant-negative manner to wild-type (WT) SRP54 and impair XBP1 splicing, leading to neutropenia and decreased size of the exocrine pancreas. Image by Veronica Bergo, created with BioRender.com.

Impaired XBP1 splicing downstream of SRP54 mutations. During physiological conditions (A), the signal recognition particle 54 (SRP54) facilitates the translocation of the ribosome-associated unspliced XBP1 (XBP1u) to the ER where it is spliced (XBP1s). In pathological conditions (B), SRP54 mutations found in patients act in a dominant-negative manner to wild-type (WT) SRP54 and impair XBP1 splicing, leading to neutropenia and decreased size of the exocrine pancreas. Image by Veronica Bergo, created with BioRender.com.

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