American Society of Hematology

Figure 1.

$Effects of N fraction–based parameters on other data reflecting ATL disease status and treatment response of aggressive ATL. (A) Representative flow cytometric plots in this study. (B) Correlation between N/P ratio and sIL-2R, LDH, and abnormal lymphocytes (/μL), colored by the status of the patients (n = 497). (C) Correlation between N fraction (%) and sIL-2R, LDH, and abnormal lymphocytes (/μL), colored by the status of the patients (n = 497). (D) Proportion of N fraction in the sample with 0% of abnormal lymphocytes. (E-I) Before and after the ratio of N/P ratio (E), N fraction (%) (F), sIL-2R (G), LDH (H), and abnormal lymphocytes (/μL) (I) at 2 different time points of the same patients (n = 121), comparison between each treatment response (PR/CR, SD, and PD). All data are shown as the mean ± standard deviation; P values were calculated by Spearman rank correlation for panels B-C or 2-tailed unpaired Student t test for panels E-I.$

Effects of N fraction–based parameters on other data reflecting ATL disease status and treatment response of aggressive ATL. (A) Representative flow cytometric plots in this study. (B) Correlation between N/P ratio and sIL-2R, LDH, and abnormal lymphocytes (/μL), colored by the status of the patients (n = 497). (C) Correlation between N fraction (%) and sIL-2R, LDH, and abnormal lymphocytes (/μL), colored by the status of the patients (n = 497). (D) Proportion of N fraction in the sample with 0% of abnormal lymphocytes. (E-I) Before and after the ratio of N/P ratio (E), N fraction (%) (F), sIL-2R (G), LDH (H), and abnormal lymphocytes (/μL) (I) at 2 different time points of the same patients (n = 121), comparison between each treatment response (PR/CR, SD, and PD). All data are shown as the mean ± standard deviation; P values were calculated by Spearman rank correlation for panels B-C or 2-tailed unpaired Student t test for panels E-I.

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