Fig. 6.
Mutations within the BCL6 exon 1 binding sites confer resistance to BCL6-mediated transrepression activity.
Responses of the 4 DLBCL-derived BSE1-mutant reporter constructs to BCL6-mediated transrepression activity were compared with those of a wild-type plasmid in cotransfection experiments in 293T cells, as described in Figure 5D.