Figure 2
Figure 2. Schematic diagram of the unfolded protein response. In response to endoplasmic reticulum (ER) stress, signals are transmitted from the ER to the cytoplasm via 3 transmembrane proteins in the ER membrane. Signaling from these 3 proteins leads to many downstream responses: Protein kinase RNA (PKR)-like ER kinase (PERK) phosphorylates elF2, which causes a global decrease in protein synthesis. IRE1 activation leads to splicing of XBP1; sXBP1 enters the nucleus and increases transcription of chaperone proteins, ER-associated protein degradation proteins, and proapoptotic proteins. ATF6 is cleaved and the cleaved portion enters the nucleus where it stimulates transcription of XBP1 and other unfolded protein response target genes.

Schematic diagram of the unfolded protein response. In response to endoplasmic reticulum (ER) stress, signals are transmitted from the ER to the cytoplasm via 3 transmembrane proteins in the ER membrane. Signaling from these 3 proteins leads to many downstream responses: Protein kinase RNA (PKR)-like ER kinase (PERK) phosphorylates elF2, which causes a global decrease in protein synthesis. IRE1 activation leads to splicing of XBP1; sXBP1 enters the nucleus and increases transcription of chaperone proteins, ER-associated protein degradation proteins, and proapoptotic proteins. ATF6 is cleaved and the cleaved portion enters the nucleus where it stimulates transcription of XBP1 and other unfolded protein response target genes.

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