Figure 3
Figure 3. Impact of genetic alterations on the tumor microenvironment. The major mechanisms of 16p13.13 gene rearrangements leading to CIITA gene fusions and 9p24.1 amplification. These mechanisms involve down-regulation of MHC class II and up-regulation of PD-1 ligands, leading to an imbalance of T-cell receptor versus costimulatory signaling. Functional consequences for the tumor microenvironment encompass induction of T-cell anergy, apoptosis, and skewed differentiation toward regulatory T cells. Orange triangles indicate chromosomal break (B). ER indicates endoplasmic reticulum.

Impact of genetic alterations on the tumor microenvironment. The major mechanisms of 16p13.13 gene rearrangements leading to CIITA gene fusions and 9p24.1 amplification. These mechanisms involve down-regulation of MHC class II and up-regulation of PD-1 ligands, leading to an imbalance of T-cell receptor versus costimulatory signaling. Functional consequences for the tumor microenvironment encompass induction of T-cell anergy, apoptosis, and skewed differentiation toward regulatory T cells. Orange triangles indicate chromosomal break (B). ER indicates endoplasmic reticulum.

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