Figure 2
Figure 2. NF-κB signaling is necessary for Tax-mediated induction of Fascin. (A) Fascin mRNA (quantitative PCR) and protein after transfection of Jurkat T cells with expression plasmids for NF-κB-deficient Tax-mutants M7 and M22 compared with wt-Tax (pcTax) and M47. The means of 3 independent experiments ± SE are shown in the upper panel. (B-C) Quantitative PCR of (B) Fascin and (C) Tax mRNA after transfection of expression plasmids for wt-Tax (20 μg) and IκBα-DN (2 μg) in Jurkat T cells. Mean values were compared using a paired t test (n = 3). **P < .01. (D) Immunoblot of Fascin after transfection of pcTax and cotransfection of pIκBα-DN or treatment with the IKK-β inhibitor ACHP solved in dimethyl sulfoxide. ACHP (10μM; 25μM) was added 24 hours after transfection for 24 hours. (E) Quantitative PCR of Fascin and 4-1BB mRNA after treatment of HTLV-1–transformed MT-2 cells and Tax-transformed Tesi cells with increasing amounts of ACHP (1, 2.5, 5, 10, and 25 μg) for 48 hours. Relative copy numbers were normalized on values obtained in dimethyl sulfoxide–treated cells.

NF-κB signaling is necessary for Tax-mediated induction of Fascin. (A) Fascin mRNA (quantitative PCR) and protein after transfection of Jurkat T cells with expression plasmids for NF-κB-deficient Tax-mutants M7 and M22 compared with wt-Tax (pcTax) and M47. The means of 3 independent experiments ± SE are shown in the upper panel. (B-C) Quantitative PCR of (B) Fascin and (C) Tax mRNA after transfection of expression plasmids for wt-Tax (20 μg) and IκBα-DN (2 μg) in Jurkat T cells. Mean values were compared using a paired t test (n = 3). **P < .01. (D) Immunoblot of Fascin after transfection of pcTax and cotransfection of pIκBα-DN or treatment with the IKK-β inhibitor ACHP solved in dimethyl sulfoxide. ACHP (10μM; 25μM) was added 24 hours after transfection for 24 hours. (E) Quantitative PCR of Fascin and 4-1BB mRNA after treatment of HTLV-1–transformed MT-2 cells and Tax-transformed Tesi cells with increasing amounts of ACHP (1, 2.5, 5, 10, and 25 μg) for 48 hours. Relative copy numbers were normalized on values obtained in dimethyl sulfoxide–treated cells.

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