Figure 4
Figure 4. ODSH and UFH but not dabigatran or rivaroxaban prevent anti-PF4/heparin Ab-induced platelet activation. Sera from patients containing anti-PF4/heparin IgG were tested in the HIPA with 0.2 IU/mL LMWH in the presence of increasing concentrations of UFH, ODSH, dabigatran, or rivaroxaban. In addition, ODSH was also tested in the presence of 0.2 IU/mL UFH. Fifteen (DMSO, n = 14) platelet donors were tested with 3 different patient sera for each anticoagulant. The figure shows the number of donors with platelet activation until 45 minutes. At a concentration of 1.04 μg/mL, ODSH started to inhibit platelet activation by anti-PF4/heparin Abs in the presence of LMWH (■), while lower concentrations of ODSH were needed in the presence of UFH (□). Dabigatran had almost no effect. The inhibition of platelet activation with high concentrations of rivaroxaban was because of its solvent carrier DMSO.

ODSH and UFH but not dabigatran or rivaroxaban prevent anti-PF4/heparinAb-induced platelet activation. Sera from patients containing anti-PF4/heparin IgG were tested in the HIPA with 0.2 IU/mL LMWH in the presence of increasing concentrations of UFH, ODSH, dabigatran, or rivaroxaban. In addition, ODSH was also tested in the presence of 0.2 IU/mL UFH. Fifteen (DMSO, n = 14) platelet donors were tested with 3 different patient sera for each anticoagulant. The figure shows the number of donors with platelet activation until 45 minutes. At a concentration of 1.04 μg/mL, ODSH started to inhibit platelet activation by anti-PF4/heparin Abs in the presence of LMWH (■), while lower concentrations of ODSH were needed in the presence of UFH (□). Dabigatran had almost no effect. The inhibition of platelet activation with high concentrations of rivaroxaban was because of its solvent carrier DMSO.

Close Modal

or Create an Account

Close Modal
Close Modal