Figure 1
Figure 1. Age at diagnosis of pediatric cases with t(8;16)(p11;p13) as compared with a pediatric AML reference cohort. Shown are two histograms depicting the age at diagnosis for two cohorts: t(8;16)(p11;p13) in dark gray, and a reference cohort of unselected pediatric AML patients treated by AML-BFM study group between 1995-2005 in light gray. (A) Age at diagnosis is shown in categories of months for the first 2 years after birth. (B) Age at diagnosis is shown in categories of 2 years until the age of 18. In t(8;16)(p11;p13) AML, after an initial peak in the occurrence shortly after birth, occurrence is stable through childhood. In the reference cohort, the frequency of congenital cases is significantly lower.

Age at diagnosis of pediatric cases with t(8;16)(p11;p13) as compared with a pediatric AML reference cohort. Shown are two histograms depicting the age at diagnosis for two cohorts: t(8;16)(p11;p13) in dark gray, and a reference cohort of unselected pediatric AML patients treated by AML-BFM study group between 1995-2005 in light gray. (A) Age at diagnosis is shown in categories of months for the first 2 years after birth. (B) Age at diagnosis is shown in categories of 2 years until the age of 18. In t(8;16)(p11;p13) AML, after an initial peak in the occurrence shortly after birth, occurrence is stable through childhood. In the reference cohort, the frequency of congenital cases is significantly lower.

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