Figure 3
Figure 3. Analysis of early cytokine secretion by HIV-1–specific CD8 T cells shows that IL-2, but not IFN-γ, distinguishes controllers from treated or untreated progressors. (A-B) Summary data of the mean magnitude of IFN-γ (A) and IL-2 (B) secretion by HIV-1–specific CD8 T cells after a 6-hour stimulation with panels of individual HLA class I–matched HIV-1 epitopes. Each column corresponds to a study participant. (C-D) Statistical analysis of mean IFN-γ (C) and IL-2 (D) early (6 hours) secretion among the 3 groups of HIV-1–infected subjects of the discovery cohort (n = 64) Mean IFN-γ responses based on HLA-I restriction in all patients. HC indicates HIV-1 controllers; CP, chronic progressors; and ARTC, ART-treated subjects. (E-F) Statistical comparison of means early IFN-γ (E) and IL-2 (F) responses to epitopes restricted by protective and nonprotective HLA-I alleles in all subjects. (G-H) Statistical analysis of early IFN-γ (G) and early IL-2 (H) responses stratified by protective versus nonprotective HLA-I alleles in HC, CP, and ARTC subjects. Throughout the figure, HC data are represented in blue, CP in red, ARTC in green, protective HLA class I alleles (B*5701/03, B*2705 and B*5801) in purple, and nonprotective HLA I alleles in orange. All comparisons of adjusted means were done using generalized linear models controlling for clustering within subjects. Vertical interval bars correspond to the SEM.

Analysis of early cytokine secretion by HIV-1–specific CD8 T cells shows that IL-2, but not IFN-γ, distinguishes controllers from treated or untreated progressors. (A-B) Summary data of the mean magnitude of IFN-γ (A) and IL-2 (B) secretion by HIV-1–specific CD8 T cells after a 6-hour stimulation with panels of individual HLA class I–matched HIV-1 epitopes. Each column corresponds to a study participant. (C-D) Statistical analysis of mean IFN-γ (C) and IL-2 (D) early (6 hours) secretion among the 3 groups of HIV-1–infected subjects of the discovery cohort (n = 64) Mean IFN-γ responses based on HLA-I restriction in all patients. HC indicates HIV-1 controllers; CP, chronic progressors; and ARTC, ART-treated subjects. (E-F) Statistical comparison of means early IFN-γ (E) and IL-2 (F) responses to epitopes restricted by protective and nonprotective HLA-I alleles in all subjects. (G-H) Statistical analysis of early IFN-γ (G) and early IL-2 (H) responses stratified by protective versus nonprotective HLA-I alleles in HC, CP, and ARTC subjects. Throughout the figure, HC data are represented in blue, CP in red, ARTC in green, protective HLA class I alleles (B*5701/03, B*2705 and B*5801) in purple, and nonprotective HLA I alleles in orange. All comparisons of adjusted means were done using generalized linear models controlling for clustering within subjects. Vertical interval bars correspond to the SEM.

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