Figure 5
Figure 5. Antibodies against CD99L2 do not affect lymphocyte transmigration in vitro and lymphocyte extravasation in vivo. (A) Transendothelial migration of SJL.PLP7 T lymphocytes is not affected by anti-CD99L2 antibodies. SJL.PLP7 cells were allowed to transmigrate through a monolayer of bEnd.5 cells grown on transwell filters for 30 minutes in the presence of 100 ng/mL SDF-1 in the lower chamber. Endothelial cells were incubated for 30 minutes before the assay either without antibodies (w/o Ab) or with preimmune control IgG (co-IgG), affinity purified anti-CD99 IgG (anti-CD99), affinity purified anti-CD99L2 IgG (anti-CD99L2), or a monoclonal antibody against ICAM-1 (anti-ICAM-1). Antibodies (each 30 μg/mL) remained present during the assay. Each panel is representative of at least 3 experiments; each measurement was done in triplicate. ***P < .001 anti-CD99 versus co-IgG. (B) Migration of lymphocytes into inflamed skin is not affected by anti-CD99L2. Radiolabeled in vivo-activated T cells were injected intravenously into hapten-challenged mice together with (50 μg each) control IgG from preimmune serum (co-IgG), or monoclonal antibodies against P-selectin and E-selectin (anti–P-/E-selectin), affinity-purified antibodies against ESAM (anti-ESAM), or affinity-purified antibodies against CD99L2 (anti-CD99L2). The bar on the left represents radioactivity accumulating in the noninflamed ear. Statistical analysis was done by Student t test. Results are representative for 3 similar experiments; for each determination, 4 mice were analyzed. Numbers on the left refer to the percentage of injected cells that were found in the analyzed ear.

Antibodies against CD99L2 do not affect lymphocyte transmigration in vitro and lymphocyte extravasation in vivo. (A) Transendothelial migration of SJL.PLP7 T lymphocytes is not affected by anti-CD99L2 antibodies. SJL.PLP7 cells were allowed to transmigrate through a monolayer of bEnd.5 cells grown on transwell filters for 30 minutes in the presence of 100 ng/mL SDF-1 in the lower chamber. Endothelial cells were incubated for 30 minutes before the assay either without antibodies (w/o Ab) or with preimmune control IgG (co-IgG), affinity purified anti-CD99 IgG (anti-CD99), affinity purified anti-CD99L2 IgG (anti-CD99L2), or a monoclonal antibody against ICAM-1 (anti-ICAM-1). Antibodies (each 30 μg/mL) remained present during the assay. Each panel is representative of at least 3 experiments; each measurement was done in triplicate. ***P < .001 anti-CD99 versus co-IgG. (B) Migration of lymphocytes into inflamed skin is not affected by anti-CD99L2. Radiolabeled in vivo-activated T cells were injected intravenously into hapten-challenged mice together with (50 μg each) control IgG from preimmune serum (co-IgG), or monoclonal antibodies against P-selectin and E-selectin (anti–P-/E-selectin), affinity-purified antibodies against ESAM (anti-ESAM), or affinity-purified antibodies against CD99L2 (anti-CD99L2). The bar on the left represents radioactivity accumulating in the noninflamed ear. Statistical analysis was done by Student t test. Results are representative for 3 similar experiments; for each determination, 4 mice were analyzed. Numbers on the left refer to the percentage of injected cells that were found in the analyzed ear.

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