Figure 1
![Figure 1. Pie chart depicting the molecular heterogeneity of CN-AML. The analysis is based on mutations in the NPM1, CEBPA, MLL, and FLT3 (ITD and tyrosine kinase domain [TKD] mutations at codons D835 and I836), NRAS, and WT1 genes. Data are derived from mutational analysis of 485 younger adult patients with CN-AML from the German AML Study Group.90](https://ash.silverchair-cdn.com/ash/content_public/journal/blood/119/2/10.1182_blood-2011-08-363291/5/zh89991183350001.jpeg?Expires=1735526373&Signature=NMXkhb5ZeD5eShlOGUHDH8H5pEEb9JdTmt7mIPlPAduPLtOIYy6tkgyRRZQRRjVJJa9v51-RJyAXTHPcDPFgmxjwCcwXZfm7sldCvoddShLLQSVZ4~jSfgz5l0Jv8LTuJTNwLXauQwcEVa0NG4QJ03BVrbVWhU-txa5DxOoU1EOalYCaQQYn75qkV~Wl-DD4RaD1O~abI2xsnoIoW8vemPPBh4pbZHCZT0V1tC8cBgqIlc-J7oPrQfHOA5ErZ~HPVWDbfmSrqXgWh97xOJ08kNGu8cqL0JGjLCE5pMpCgjGGNSp8AZai9~GD-TPqLzkKjxHGtEjLWbeu4v1wH6fmSw__&Key-Pair-Id=APKAIE5G5CRDK6RD3PGA)
Pie chart depicting the molecular heterogeneity of CN-AML. The analysis is based on mutations in the NPM1, CEBPA, MLL, and FLT3 (ITD and tyrosine kinase domain [TKD] mutations at codons D835 and I836), NRAS, and WT1 genes. Data are derived from mutational analysis of 485 younger adult patients with CN-AML from the German AML Study Group.90
