Fig. 6.
Fig. 6. IL-8, G-CSF, and MCP1 are not responsible for FucS-induced mobilization. / (A) CFCs in PB of wild-type B6/129 controls (n = 4) or GCSFR−/− mice (n = 9) before (open bars) or after one intravenous injection of 100 mg/kg FucS (closed bars). Differences were statistically significant by the Student t test between untreated and treated (P < .0001) in both groups. (B) Total numbers of WBCs (circles) and CFCs (bars) in PB of wild-type B6/129 controls (n = 4) or MCP1−/− mice (n = 5) before (open circles and bars) or after one intravenous injection of 100 mg/kg FucS (closed circles and bars). Error bars represent the mean ± SEM. Differences were statistically significant by the Student t test between untreated and treated for both CFCs and WBCs (P < .0001) in both groups but not between wild-type and MCP1−/−.

IL-8, G-CSF, and MCP1 are not responsible for FucS-induced mobilization.

(A) CFCs in PB of wild-type B6/129 controls (n = 4) or GCSFR−/− mice (n = 9) before (open bars) or after one intravenous injection of 100 mg/kg FucS (closed bars). Differences were statistically significant by the Student t test between untreated and treated (P < .0001) in both groups. (B) Total numbers of WBCs (circles) and CFCs (bars) in PB of wild-type B6/129 controls (n = 4) or MCP1−/− mice (n = 5) before (open circles and bars) or after one intravenous injection of 100 mg/kg FucS (closed circles and bars). Error bars represent the mean ± SEM. Differences were statistically significant by the Student t test between untreated and treated for both CFCs and WBCs (P < .0001) in both groups but not between wild-type and MCP1−/−.

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