American Society of Hematology

Fig. 4.

$Fig. 4. Time course correlating the exposure of phosphatidylserine by cells during cell death with enhanced binding of FITC-heparin. / Jurkat cells were treated with Camptothecin, and samples were taken at timed intervals for labeling with PE–annexin V and FITC-heparin (500 nM). The results show the correlation between the fraction of cells with exposed PS (enhanced PE–annexin V binding ▴-▴) to the fraction of cells binding FITC-heparin with an RFI that was 10-fold higher than untreated cells (▪-▪). Counted cells are expressed as a percentage of PE–annexin V–positive or FITC-heparin–positive cells in the 24-hour sample.$

Time course correlating the exposure of phosphatidylserine by cells during cell death with enhanced binding of FITC-heparin.

Jurkat cells were treated with Camptothecin, and samples were taken at timed intervals for labeling with PE–annexin V and FITC-heparin (500 nM). The results show the correlation between the fraction of cells with exposed PS (enhanced PE–annexin V binding ▴-▴) to the fraction of cells binding FITC-heparin with an RFI that was 10-fold higher than untreated cells (▪-▪). Counted cells are expressed as a percentage of PE–annexin V–positive or FITC-heparin–positive cells in the 24-hour sample.

This Feature Is Available To Subscribers Only