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Journal Articles

Hypoxia-Adapted CML Cells Are More Primitive Population and Are Eradicated by Glyoxalase-1 Inhibitors.

Miki Takeuchi, MD, Shinya Kimura, MD, PhD, Junya Kuroda, MD, PhD, Eishi Ashihara, MD, PhD, Makoto Kawatani, PhD, Hiroyuki Osada, PhD, Kazuo Umezawa, PhD, Eiko Yasui, PhD, Masaya Imoto, PhD, Takashi Tsuruo, MD, PhD, Asumi Yokota, MMSc, Ruriko Tanaka, MMSc, Rina Nagao, Tatsutoshi Nakahata, M.D., Ph.D., Yoshihide Fujiyama, MD, PhD, Taira Maekawa, MD, PhD

Blood (2009) 114 (22): 2166.

https://doi.org/10.1182/blood.V114.22.2166.2166

Published: 2009

Journal Articles

Inhibition of Wnt/β-Catenin Signaling by AV65 Treatment Caused Cell Cycle Arrest and Induced Caspase-Independent or -Dependent Apoptosis in CML Cells.

Rina Nagao, Eishi Ashihara, MD, PhD, Shinya Kimura, MD, PhD, Hisayuki Yao, MD, Miki Takeuchi, MD, Asumi Yokota, MMSc, Ruriko Tanaka, MMSc, Hideyo Hirai, MD, PhD, Jeffrey W Strovel, Janak Padia, Wieslaw M Cholody, Taira Maekawa, MD, PhD

Blood (2009) 114 (22): 2184.

https://doi.org/10.1182/blood.V114.22.2184.2184

Published: 2009

Journal Articles

Automated and Rapid Detection of BCR-ABL Kinase Domain Mutations in IM Resistant Patients with Ph+ Leukemias.

Ruriko Tanaka, MMSc, Shinya Kimura, MD, PhD, Toshiya Hosomi, Mitsuharu Hirai, Rina Nagao, Asumi Yokota, MMSc, Yuri Kamitsuji, MD, PhD, Eri Kawata, MD, PhD, Hisayuki Yao, MD, Kazuki Sakai, MD, Hideyo Hirai, MD, PhD, Eishi Ashihara, MD, PhD, Souichi Adachi, MD, PhD, Taira Maekawa, MD, PhD

Blood (2009) 114 (22): 2590.

https://doi.org/10.1182/blood.V114.22.2590.2590

Published: 2009

Journal Articles

Osteoclasts Are Involved in the Maintenance of Dormant Leukemic Cells.

Asumi Yokota, MMSc, Shinya Kimura, MD, PhD, Ruriko Tanaka, MMSc, Miki Takeuchi, MD, Hisayuki Yao, MD, Kazuki Sakai, MD, Rina Nagao, Eishi Ashihara, MD, PhD, Taira Maekawa, MD, PhD

Blood (2009) 114 (22): 4245.

https://doi.org/10.1182/blood.V114.22.4245.4245

Published: 2009

Journal Articles

A Novel β-catenin Inhibitor, AV65 Suppresses the Growth of CML Cell Lines Which Acquire Imatinib-Resistance Because of Abl Kinase Domain Mutations Including T315I and Hypoxia-Adaptation.

Rina Nagao, Shinya Kimura, MD, PhD, Eishi Ashihara, MD, PhD, Miki Takeuchi, MD, Ruriko Tanaka, MMSc, Asumi Yokota, MMSc, Hisayuki Yao, MD, Jeffrey W Strovel, Wieslaw M Cholody, Taira Maekawa, MD, PhD

Blood (2008) 112 (11): 1081.

https://doi.org/10.1182/blood.V112.11.1081.1081

Published: 2008

Journal Articles

Activity of the Multi-Targeted Kinase Inhibitor, AT9283 on Imatinib-Resistant CML Models.

Ruriko Tanaka, MMSc, Matthew S Squires, PhD, Shinya Kimura, MD, PhD, Asumi Yokota, MMSc, Kirsty Mallett, PhD, Tomoko Smyth, PhD, Neil T Thompson, PhD, Rina Nagao, Takahiro Yamauchi, MD, Takashi Ushiki, MD, John F Lyons, PhD, Taira Maekawa, MD, PhD

Blood (2008) 112 (11): 1104.

https://doi.org/10.1182/blood.V112.11.1104.1104

Published: 2008

Journal Articles

Roles of Osteoclasts in Leukemic Cells

Asumi Yokota, MMSc, Shinya Kimura, MD, PhD, Ruriko Tanaka, MMSc, Rina Nagao, Kazuki Sakai, MD, Hisayuki Yao, MD, Miki Takeuchi, MD, Makoto Kawatani, PhD, Hiroyuki Osada, PhD, Eishi Ashihara, MD, PhD, Taira Maekawa, MD, PhD

Blood (2008) 112 (11): 3184.

https://doi.org/10.1182/blood.V112.11.3184.3184

Published: 2008

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Hypoxia-Adapted CML Cells Are More Primitive Population and Are Eradicated by Glyoxalase-1 Inhibitors.

Inhibition of Wnt/β-Catenin Signaling by AV65 Treatment Caused Cell Cycle Arrest and Induced Caspase-Independent or -Dependent Apoptosis in CML Cells.

Automated and Rapid Detection of BCR-ABL Kinase Domain Mutations in IM Resistant Patients with Ph+ Leukemias.

Osteoclasts Are Involved in the Maintenance of Dormant Leukemic Cells.

A Novel β-catenin Inhibitor, AV65 Suppresses the Growth of CML Cell Lines Which Acquire Imatinib-Resistance Because of Abl Kinase Domain Mutations Including T315I and Hypoxia-Adaptation.

Activity of the Multi-Targeted Kinase Inhibitor, AT9283 on Imatinib-Resistant CML Models.

Roles of Osteoclasts in Leukemic Cells

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ASH Publications

American Society of Hematology

Hypoxia-Adapted CML Cells Are More Primitive Population and Are Eradicated by Glyoxalase-1 Inhibitors.

Inhibition of Wnt/β-Catenin Signaling by AV65 Treatment Caused Cell Cycle Arrest and Induced Caspase-Independent or -Dependent Apoptosis in CML Cells.

Automated and Rapid Detection of BCR-ABL Kinase Domain Mutations in IM Resistant Patients with Ph+ Leukemias.

Osteoclasts Are Involved in the Maintenance of Dormant Leukemic Cells.

A Novel β-catenin Inhibitor, AV65 Suppresses the Growth of CML Cell Lines Which Acquire Imatinib-Resistance Because of Abl Kinase Domain Mutations Including T315I and Hypoxia-Adaptation.

Activity of the Multi-Targeted Kinase Inhibitor, AT9283 on Imatinib-Resistant CML Models.

Roles of Osteoclasts in Leukemic Cells

Email alerts

ASH Publications

American Society of Hematology

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