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Journal Articles

Hemolytic uremic syndrome–associated Shiga toxins promote endothelial-cell secretion and impair ADAMTS13 cleavage of unusually large von Willebrand factor multimers

Leticia H. Nolasco, Nancy A. Turner, Aubrey Bernardo, Zhenyin Tao, Thomas G. Cleary, Jing-fei Dong, Joel L. Moake

Blood (2005) 106 (13): 4199–4209.

https://doi.org/10.1182/blood-2005-05-2111

Published: 2005

Journal Articles

Cleavage of ultralarge multimers of von Willebrand factor by C -terminal–truncated mutants of ADAMTS-13 under flow

Zhenyin Tao, Yongtao Wang, Huiwei Choi, Aubrey Bernardo, Kenji Nishio, J. Evan Sadler, José A. López, Jing-fei Dong

Blood (2005) 106 (1): 141–143.

https://doi.org/10.1182/blood-2004-11-4188

Published: 2005

Journal Articles

Effects of inflammatory cytokines on the release and cleavage of the endothelial cell–derived ultralarge von Willebrand factor multimers under flow

Aubrey Bernardo, Chalmette Ball, Leticia Nolasco, Joel F. Moake, Jing-fei Dong

Blood (2004) 104 (1): 100–106.

https://doi.org/10.1182/blood-2004-01-0107

Published: 2004

Journal Articles

P-selectin anchors newly released ultralarge von Willebrand factor multimers to the endothelial cell surface

Arnoldo Padilla, Joel L. Moake, Aubrey Bernardo, Chalmette Ball, Yongtao Wang, Maneesh Arya, Leticia Nolasco, Nancy Turner, Michael C. Berndt, Bahman Anvari, José A. López, Jing-Fei Dong

Blood (2004) 103 (6): 2150–2156.

https://doi.org/10.1182/blood-2003-08-2956

Published: 2004

Journal Articles

ADAMTS-13 rapidly cleaves newly secreted ultralarge von Willebrand factor multimers on the endothelial surface under flowing conditions

Jing-fei Dong, Joel L. Moake, Leticia Nolasco, Aubrey Bernardo, Wendy Arceneaux, Corie N. Shrimpton, Alicia J. Schade, Larry V. McIntire, Kazuo Fujikawa, José A. López

Blood (2002) 100 (12): 4033–4039.

https://doi.org/10.1182/blood-2002-05-1401

Published: 2002

NARROW

Hemolytic uremic syndrome–associated Shiga toxins promote endothelial-cell secretion and impair ADAMTS13 cleavage of unusually large von Willebrand factor multimers

Cleavage of ultralarge multimers of von Willebrand factor by C -terminal–truncated mutants of ADAMTS-13 under flow

Effects of inflammatory cytokines on the release and cleavage of the endothelial cell–derived ultralarge von Willebrand factor multimers under flow

P-selectin anchors newly released ultralarge von Willebrand factor multimers to the endothelial cell surface

ADAMTS-13 rapidly cleaves newly secreted ultralarge von Willebrand factor multimers on the endothelial surface under flowing conditions

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ASH Publications

American Society of Hematology

Hemolytic uremic syndrome–associated Shiga toxins promote endothelial-cell secretion and impair ADAMTS13 cleavage of unusually large von Willebrand factor multimers

Cleavage of ultralarge multimers of von Willebrand factor by C -terminal–truncated mutants of ADAMTS-13 under flow

Effects of inflammatory cytokines on the release and cleavage of the endothelial cell–derived ultralarge von Willebrand factor multimers under flow

P-selectin anchors newly released ultralarge von Willebrand factor multimers to the endothelial cell surface

ADAMTS-13 rapidly cleaves newly secreted ultralarge von Willebrand factor multimers on the endothelial surface under flowing conditions

Email alerts

ASH Publications

American Society of Hematology

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