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A Preliminary Analysis of FLAM (Italian Non-Interventional Multi-center Study of FLT3 mutated AML patients): FLT3 Receptor Gene Mutational Analysis and FLT3 Inhibitors Administration in the Real-Life Clinical Practice

Blood (2020) 136 (Supplement 1): 32–34.

https://doi.org/10.1182/blood-2020-139981

Published: 2020

Journal Articles

Higher Monocyte Chemoattractant Protein-1 Levels in Myelofibrosis Are Sustained By the rs1024611 Single Nucleotide Polymorphism and Correlate with Disease Subtype and Severity

Elena Masselli, MD, Giulia Pozzi, MS, Cecilia Carubbi, PhD, Luca Pagliaro, MD, Elena Follini, MD, Benedetta Cambò, MD, Monica Crugnola, MD, Giuliana Gobbi, PhD, Prisco Mirandola, PhD, Franco Aversa, MD, Marco Vitale, MD

Blood (2018) 132 (Supplement 1): 1785.

https://doi.org/10.1182/blood-2018-99-111719

Published: 2018

Journal Articles

Cell-Type Specific Mechanisms of Hematopoietic Stem Cell (HSC) Expansion Underpin Progressive Disease in Myelodysplastic Syndromes (MDS) and Provide a Rationale for Targeted Therapies

Blood (2018) 132 (Supplement 1): 1798.

https://doi.org/10.1182/blood-2018-99-116881

Published: 2018

Journal Articles

The -2518A/G (rs1026411) Regulatory Region Polymorphism of the Monocyte Chemoattractant Protein-1 (MCP-1) Is Associated with Secondary Myelofibrosis

Elena Masselli, MD, Cecilia Carubbi, PhD, Benedetta Cambò, MD, Giulia Pozzi, MS, Giuliana Gobbi, PhD, Prisco Mirandola, PhD, Elena Follini, MD, Luca Pagliaro, MD, Francesco Bonatti, PhD, Antonio Percesepe, MD PhD, Franco Aversa, MD, Marco Vitale, MD

Blood (2017) 130 (Supplement 1): 5286.

https://doi.org/10.1182/blood.V130.Suppl_1.5286.5286

Published: 2017

Journal Articles

Bone Marrow Remodelling and Topographic Redistribution of CD34 Positive Progenitors Characterize the Progression of Myelodysplastic Syndromes and Their Evolution to AML

Federico Quaini, MD PhD, Rodanthi Fioretzaki, MD, Monica Crugnola, MD, Angela Falco, PhD, Denise Madeddu, PhD, Luisa Craviotto, MD, Benedetta Cambò, MD, Lucia Prezioso, MD PhD, Gabriella Sammarelli, PhD, Giannalisa Todaro, PhD, Gallia Graiani, PhD, Cristina Mancini, MD, Eugenia Martella, MD, Francesca Re, MD, Nicola Giuliani, MD, PhD, Franco Aversa, MD

Blood (2016) 128 (22): 4302.

https://doi.org/10.1182/blood.V128.22.4302.4302

Published: 2016

Journal Articles

Therapeutic Strategy for Elderly Patients with ACUTE Myeloid Leukemia (AML) Carrying the DNMT3A Mutation

Elena Rossetti, MD, Monica Crugnola, MD, Cecilia Caramatti, MD, Luisa Craviotto, MD, Elena Masselli, MD, Lucia Prezioso, MD PhD, Alessandro Monti, MD, Benedetta Cambò, MD, Chiara Lambertini, MD, Claudio Pietropaolo, Anna Minardi, Gabriella Sammarelli, PhD, Sabrina Bonomini, Rodanthi Fioretzaki, MD, Franco Aversa, MD

Blood (2014) 124 (21): 5269.

https://doi.org/10.1182/blood.V124.21.5269.5269

Published: 2014

Journal Articles

Protein Kinase C Epsilon Inhibition Restores In-Vitro Megakaryocyte Differentiation Of Primary Myelofibrosis Hematopoietic Progenitors

Elena Masselli, MD, Prisco Mirandola, PhD, Giuliana Gobbi, PhD, Cecilia Carubbi, MS, Sabrina Bonomini, PhD, Monica Crugnola, MD, Filomena Russo, MD, Luisa Craviotto, MD, Benedetta Cambò, MD, Caterina Plenteda, MD, Elena Rossetti, MD, Cecilia Caramatti, MD, Franco Aversa, MD, Marco Vitale, MD

Blood (2013) 122 (21): 114.

https://doi.org/10.1182/blood.V122.21.114.114

Published: 2013

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A Preliminary Analysis of FLAM (Italian Non-Interventional Multi-center Study of FLT3 mutated AML patients): FLT3 Receptor Gene Mutational Analysis and FLT3 Inhibitors Administration in the Real-Life Clinical Practice

Higher Monocyte Chemoattractant Protein-1 Levels in Myelofibrosis Are Sustained By the rs1024611 Single Nucleotide Polymorphism and Correlate with Disease Subtype and Severity

Cell-Type Specific Mechanisms of Hematopoietic Stem Cell (HSC) Expansion Underpin Progressive Disease in Myelodysplastic Syndromes (MDS) and Provide a Rationale for Targeted Therapies

The -2518A/G (rs1026411) Regulatory Region Polymorphism of the Monocyte Chemoattractant Protein-1 (MCP-1) Is Associated with Secondary Myelofibrosis

Bone Marrow Remodelling and Topographic Redistribution of CD34 Positive Progenitors Characterize the Progression of Myelodysplastic Syndromes and Their Evolution to AML

Therapeutic Strategy for Elderly Patients with ACUTE Myeloid Leukemia (AML) Carrying the DNMT3A Mutation

Protein Kinase C Epsilon Inhibition Restores In-Vitro Megakaryocyte Differentiation Of Primary Myelofibrosis Hematopoietic Progenitors

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ASH Publications

American Society of Hematology

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A Preliminary Analysis of FLAM (Italian Non-Interventional Multi-center Study of FLT3 mutated AML patients): FLT3 Receptor Gene Mutational Analysis and FLT3 Inhibitors Administration in the Real-Life Clinical Practice

Higher Monocyte Chemoattractant Protein-1 Levels in Myelofibrosis Are Sustained By the rs1024611 Single Nucleotide Polymorphism and Correlate with Disease Subtype and Severity

Cell-Type Specific Mechanisms of Hematopoietic Stem Cell (HSC) Expansion Underpin Progressive Disease in Myelodysplastic Syndromes (MDS) and Provide a Rationale for Targeted Therapies

The -2518A/G (rs1026411) Regulatory Region Polymorphism of the Monocyte Chemoattractant Protein-1 (MCP-1) Is Associated with Secondary Myelofibrosis

Bone Marrow Remodelling and Topographic Redistribution of CD34 Positive Progenitors Characterize the Progression of Myelodysplastic Syndromes and Their Evolution to AML

Therapeutic Strategy for Elderly Patients with ACUTE Myeloid Leukemia (AML) Carrying the DNMT3A Mutation

Protein Kinase C Epsilon Inhibition Restores In-Vitro Megakaryocyte Differentiation Of Primary Myelofibrosis Hematopoietic Progenitors

Email alerts

ASH Publications

American Society of Hematology

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