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Journal Articles

Activation of Notch-Hes Signaling Pathway Impairs Osteogenic Differentiation and Hematopoietic Stem Cell-Supporting Capacitiy of Mesenchymal Stromal Cells Derived from Myelodysplastic Syndromes Patients

Chunkang Chang, MDPhD, Chengming Fei, MD, Juan Guo, MD, Youshan Zhao, MD, Shucheng Gu, MD, Xiao Li, MD PhD

Blood (2015) 126 (23): 2857.

https://doi.org/10.1182/blood.V126.23.2857.2857

Published: 2015

Journal Articles

A Primary Study of the Gene Mutations in Predicting Treatment Response to Decitabine in Patients with MDS

Chunkang Chang, MDPhD, Youshan Zhao, MD, Feng Xu, Xiao Li, MD PhD

Blood (2015) 126 (23): 1689.

https://doi.org/10.1182/blood.V126.23.1689.1689

Published: 2015

Journal Articles

Labile Plasma Iron (LPI), More Practical and More Sensitive to Iron Overload in Myelodysplastic Syndromes

Chunkang Chang, MDPhD, Shucheng Gu, MD, Juan Guo, MD, Youshan Zhao, MD, Chengming Fei, MD, Xiao Li, MD PhD

Blood (2015) 126 (23): 5230.

https://doi.org/10.1182/blood.V126.23.5230.5230

Published: 2015

Journal Articles

Chidamide, a Novel Histone Deacetylase Inhibitor, Displays Potent Antitumor Activity Against MDS Cells Mainly through JAK2/STAT3 Signaling Inhibition

Chunkang Chang, MDPhD, Sida Zhao, MD, Juan Guo, MD, Youshan Zhao, MD, Chengming Fei, MD, Shucheng Gu, MD, Xiao Li, MD PhD

Blood (2015) 126 (23): 5233.

https://doi.org/10.1182/blood.V126.23.5233.5233

Published: 2015

Journal Articles

DHX9 Mutations Are Identified As a Novel Recurrent Event in Patients with Myelodysplastic Syndromes and Closely Related to Bone Marrow Failure

Wenhui Shi, Feng Xu, Chunkang Chang, MDPhD, Xiao Li, MD PhD

Blood (2015) 126 (23): 1651.

https://doi.org/10.1182/blood.V126.23.1651.1651

Published: 2015

Journal Articles

EZH2-Mediated Activation of Serine Biosynthetic Pathway Is Critical for Resistance of Clonal Cells to Stress-Related Apoptosis in Low-Grade Myelodysplastic Syndrome

Feng Xu, Chunkang Chang, MDPhD, Wenhui Shi, Xiao Li, MD PhD

Blood (2015) 126 (23): 1660.

https://doi.org/10.1182/blood.V126.23.1660.1660

Published: 2015

NARROW

Activation of Notch-Hes Signaling Pathway Impairs Osteogenic Differentiation and Hematopoietic Stem Cell-Supporting Capacitiy of Mesenchymal Stromal Cells Derived from Myelodysplastic Syndromes Patients

A Primary Study of the Gene Mutations in Predicting Treatment Response to Decitabine in Patients with MDS

Labile Plasma Iron (LPI), More Practical and More Sensitive to Iron Overload in Myelodysplastic Syndromes

Chidamide, a Novel Histone Deacetylase Inhibitor, Displays Potent Antitumor Activity Against MDS Cells Mainly through JAK2/STAT3 Signaling Inhibition

DHX9 Mutations Are Identified As a Novel Recurrent Event in Patients with Myelodysplastic Syndromes and Closely Related to Bone Marrow Failure

EZH2-Mediated Activation of Serine Biosynthetic Pathway Is Critical for Resistance of Clonal Cells to Stress-Related Apoptosis in Low-Grade Myelodysplastic Syndrome

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ASH Publications

American Society of Hematology

Activation of Notch-Hes Signaling Pathway Impairs Osteogenic Differentiation and Hematopoietic Stem Cell-Supporting Capacitiy of Mesenchymal Stromal Cells Derived from Myelodysplastic Syndromes Patients

A Primary Study of the Gene Mutations in Predicting Treatment Response to Decitabine in Patients with MDS

Labile Plasma Iron (LPI), More Practical and More Sensitive to Iron Overload in Myelodysplastic Syndromes

Chidamide, a Novel Histone Deacetylase Inhibitor, Displays Potent Antitumor Activity Against MDS Cells Mainly through JAK2/STAT3 Signaling Inhibition

DHX9 Mutations Are Identified As a Novel Recurrent Event in Patients with Myelodysplastic Syndromes and Closely Related to Bone Marrow Failure

EZH2-Mediated Activation of Serine Biosynthetic Pathway Is Critical for Resistance of Clonal Cells to Stress-Related Apoptosis in Low-Grade Myelodysplastic Syndrome

Email alerts

ASH Publications

American Society of Hematology

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