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Journal Articles

PU.1 Promotes Monocyte Development in Part By Transcriptionally Activating c-Fos, Egr-1 and Egr-2

Hafiza Sidra Bashir, Binod G C, Fan Dong

Blood (2024) 144 (Supplement 1): 1152.

https://doi.org/10.1182/blood-2024-203383

Published: 2024

Journal Articles

Bcl-Xl Is Important for the Antiapoptotic Activity of Gfi1 and Is Upregulated By Gfi1 through Hemgn

Binod G C, Fan Dong

Blood (2024) 144 (Supplement 1): 5624.

https://doi.org/10.1182/blood-2024-207382

Published: 2024

Journal Articles

Hemgn Upregulation By Gfi1 through PU.1 Repression Contributes to Gfi1's Anti-Apoptotic Activity

Binod G C, Fan Dong

Blood (2024) 144 (Supplement 1): 5619.

https://doi.org/10.1182/blood-2024-198668

Published: 2024

Journal Articles

Gfi1 Upregulates c-Myc Expression and Promotes c-Myc-Driven Cell Proliferation

Yangyang Zhang, MD, Fan Dong, MD PhD

Blood (2019) 134 (Supplement_1): 3769.

https://doi.org/10.1182/blood-2019-124833

Published: 2019

Journal Articles

Gfi1-Mediated Repression of C-Fos , Egr1 and Egr2 , and Inhibition of ERK1/2 Signaling Contribute to the Role of Gfi1 in Granulopoiesis

Yangyang Zhang, MD, Nan Hu, PhD, Fan Dong, MD PhD

Blood (2017) 130 (Supplement 1): 3765.

https://doi.org/10.1182/blood.V130.Suppl_1.3765.3765

Published: 2017

Journal Articles

The Duration of Erk1/2 Signaling Determines Neutrophil Versus Monocyte Cell Fates in Response to G-CSF and M-CSF

Nan Hu, MS, Yaling Qiu, MD, Fan Dong, MD, PhD

Blood (2014) 124 (21): 4353.

https://doi.org/10.1182/blood.V124.21.4353.4353

Published: 2014

Journal Articles

A Carboxy-Terminally Truncated G-CSF Receptor Abolishes Apoptosis Induction By Neutrophil Elastase G185R Mutant In Myeloid Cells,

Fan Dong, M.D., Ph.D., Yaling Qiu, M.D., Nan Hu, Alan D. Friedman, M.D.

Blood (2013) 122 (21): 443.

https://doi.org/10.1182/blood.V122.21.443.443

Published: 2013

Journal Articles

Gfi1 Is Repressed by p53 and Inhibits DNA Damage-Induced Apoptosis

Pei Du, Fangqiang Tang, Ph.D., Yaling Qiu, M.D., Fan Dong, M.D., Ph.D.

Blood (2012) 120 (21): 1228.

https://doi.org/10.1182/blood.V120.21.1228.1228

Published: 2012

Journal Articles

Mouse models of MYH9 -related disease: mutations in nonmuscle myosin II-A

Yingfan Zhang, Mary Anne Conti, Daniela Malide, Fan Dong, Aibing Wang, Yelena A. Shmist, Chengyu Liu, Patricia Zerfas, Mathew P. Daniels, Chi-Chao Chan, Elliot Kozin, Bechara Kachar, Michael J. Kelley, Jeffrey B. Kopp, Robert S. Adelstein

Blood (2012) 119 (1): 238–250.

https://doi.org/10.1182/blood-2011-06-358853

Published: 2012

Includes: Multimedia, Supplemental data

Journal Articles

Gfi-1 Represses CDKN2B Encoding p15 INK4B through Interaction with Miz-1

Qingquan Liu, Suchitra Basu, Yaling Qiu, Fan Dong

Blood (2008) 112 (11): 3578.

https://doi.org/10.1182/blood.V112.11.3578.3578

Published: 2008

Journal Articles

The C-Terminal Region of the G-CSF Receptor Is Required for Induction of Neutrophil Elastase Expression in Myeloid Cells: Implication for Understanding CSF3R Mutations in Acute Myeloid Leukemia Evolving from Severe Congenital Neutropenia.

Fan Dong, MD, PhD, Yaling Qiu, M.D., Alan D. Friedman, Ph.D., Qingquan Liu

Blood (2006) 108 (11): 1154.

https://doi.org/10.1182/blood.V108.11.1154.1154

Published: 2006

Journal Articles

Functional Gfi-1 Is Important for BCR/ABL-Mediated Proliferation and Survival of Myeloid Cells.

Suchitra Basu, Yaling Qiu, M.D., Fan Dong, M.D., Ph.D.

Blood (2006) 108 (11): 4804.

https://doi.org/10.1182/blood.V108.11.4804.4804

Published: 2006

Journal Articles

Gfi-1 Represses C/EBPe and Is Required for the Survival and Proliferation of Myeloid Cells Stimulated by G-CSF.

Fan Dong, MD, PhD, Dazhong Zhuang, PhD, Yaling Qiu, MD

Blood (2005) 106 (11): 3153.

https://doi.org/10.1182/blood.V106.11.3153.3153

Published: 2005

Journal Articles

Expression of C/EBPε Restores G-CSF-Induced Granulocytic Differentiation of Myeloid Cells Expressing the Carboxy Truncated G-CSF Receptor Associated with AML/SCN.

Fan Dong, MD, PhD, Dazhong Zhuang, PhD, Yaling Qiu, MD

Blood (2005) 106 (11): 3154.

https://doi.org/10.1182/blood.V106.11.3154.3154

Published: 2005

Journal Articles

Rod mutations associated with MYH9- related disorders disrupt nonmuscle myosin-IIA assembly

Josef D. Franke, Fan Dong, Wayne L. Rickoll, Michael J. Kelley, Daniel P. Kiehart

Blood (2005) 105 (1): 161–169.

https://doi.org/10.1182/blood-2004-06-2067

Published: 2005

Journal Articles

A Serine Protease Activity Is Induced by G-CSF and Degrades Stat5 Proteins in Myeloid Cells.

Yaling Qiu, M.D., Dazhong Zhuang, Ph.D., Alexandra MacRae, Fan Dong, M.D., Ph.D.

Blood (2004) 104 (11): 2180.

https://doi.org/10.1182/blood.V104.11.2180.2180

Published: 2004

Journal Articles

Involvement of PKCe in the Negative Regulation of Akt Activation Stimulated by G-CSF.

Hong Liu, M.D., Ph.D., Fan Dong, M.D., Ph.D.

Blood (2004) 104 (11): 2187.

https://doi.org/10.1182/blood.V104.11.2187.2187

Published: 2004

Journal Articles

Activation of Akt kinase by granulocyte colony-stimulating factor (G-CSF): evidence for the role of a tyrosine kinase activity distinct from the janus kinases

Fan Dong, Andrew C. Larner

Blood (2000) 95 (5): 1656–1662.

https://doi.org/10.1182/blood.V95.5.1656.005k29_1656_1662

Published: 2000

Journal Articles

Prolonged STAT1 Activation Related to the Growth Arrest of Malignant Lymphoma Cells by Interferon-α

Philip M. Grimley, Hui Fang, Hallgeir Rui, Emanuel F. Petricoin III, Subhransu Ray, Fan Dong, Karen H. Fields, Renqiu Hu, Kathryn C. Zoon, Susette Audet, Judy Beeler

Blood (1998) 91 (8): 3017–3027.

https://doi.org/10.1182/blood.V91.8.3017.3017_3017_3027

Published: 1998

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PU.1 Promotes Monocyte Development in Part By Transcriptionally Activating c-Fos, Egr-1 and Egr-2

Bcl-Xl Is Important for the Antiapoptotic Activity of Gfi1 and Is Upregulated By Gfi1 through Hemgn

Hemgn Upregulation By Gfi1 through PU.1 Repression Contributes to Gfi1's Anti-Apoptotic Activity

Gfi1 Upregulates c-Myc Expression and Promotes c-Myc-Driven Cell Proliferation

Gfi1-Mediated Repression of C-Fos , Egr1 and Egr2 , and Inhibition of ERK1/2 Signaling Contribute to the Role of Gfi1 in Granulopoiesis

The Duration of Erk1/2 Signaling Determines Neutrophil Versus Monocyte Cell Fates in Response to G-CSF and M-CSF

A Carboxy-Terminally Truncated G-CSF Receptor Abolishes Apoptosis Induction By Neutrophil Elastase G185R Mutant In Myeloid Cells,

Gfi1 Is Repressed by p53 and Inhibits DNA Damage-Induced Apoptosis

Mouse models of MYH9 -related disease: mutations in nonmuscle myosin II-A

Gfi-1 Represses CDKN2B Encoding p15 INK4B through Interaction with Miz-1

The C-Terminal Region of the G-CSF Receptor Is Required for Induction of Neutrophil Elastase Expression in Myeloid Cells: Implication for Understanding CSF3R Mutations in Acute Myeloid Leukemia Evolving from Severe Congenital Neutropenia.

Functional Gfi-1 Is Important for BCR/ABL-Mediated Proliferation and Survival of Myeloid Cells.

Gfi-1 Represses C/EBPe and Is Required for the Survival and Proliferation of Myeloid Cells Stimulated by G-CSF.

Expression of C/EBPε Restores G-CSF-Induced Granulocytic Differentiation of Myeloid Cells Expressing the Carboxy Truncated G-CSF Receptor Associated with AML/SCN.

Rod mutations associated with MYH9- related disorders disrupt nonmuscle myosin-IIA assembly

A Serine Protease Activity Is Induced by G-CSF and Degrades Stat5 Proteins in Myeloid Cells.

Involvement of PKCe in the Negative Regulation of Akt Activation Stimulated by G-CSF.

Activation of Akt kinase by granulocyte colony-stimulating factor (G-CSF): evidence for the role of a tyrosine kinase activity distinct from the janus kinases

Prolonged STAT1 Activation Related to the Growth Arrest of Malignant Lymphoma Cells by Interferon-α

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