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Journal Articles

Understanding the Transferrin Receptor and Cellular Iron Deficiency Outside the Erythron

Nancy C. Andrews, MD PhD

Blood (2017) 130 (Supplement 1): SCI-42.

https://doi.org/10.1182/blood.V130.Suppl_1.SCI-42.SCI-42

Published: 2017

Journal Articles

Genetic Loss of Tmprss6 Increases Effective Erythropoiesis in a Mouse Model of β-Thalassemia

David B. Stagg, BS, Sara Gardenghi, PhD, Stefano Rivella, PhD, Nancy C. Andrews, MD, PhD, Karin E. Finberg, MD, PhD

Blood (2012) 120 (21): 482.

https://doi.org/10.1182/blood.V120.21.482.482

Published: 2012

Journal Articles

The Serine Protease Tmprss6 Regulates Hepcidin Expression, but Its Loss Does Not Cause Systemic Iron Deficiency In the Fetal and Neonatal Periods

Ramsey M. Wehbe, B.S., Rebecca L. Whittlesey, B.S., Nancy C. Andrews, MD, PhD, Karin E. Finberg, MD, PhD

Blood (2010) 116 (21): 4258.

https://doi.org/10.1182/blood.V116.21.4258.4258

Published: 2010

Journal Articles

Tmprss6, An Inhibitor of Hepatic Bmp/Smad Signaling, Is Required for Hepcidin Suppression and Iron Loading In a Mouse Model of β-Thalassemia

Karin E. Finberg, MD, PhD, Rebecca L. Whittlesey, BS, Stefano Rivella, PhD, Nancy C. Andrews, MD, PhD

Blood (2010) 116 (21): 164.

https://doi.org/10.1182/blood.V116.21.164.164

Published: 2010

Journal Articles

Hepcidin as a Therapeutic Tool to Limit Iron Overload and Improve Anemia In β-Thalassemia

Sara Gardenghi, PhD, Pedro Ramos, Cindy N. Roy, PhD, Nancy C. Andrews, MD, PhD, Elizabeta Nemeth, PhD, Xiuli An, MD, PhD, Mohandas Narla, DSc, Yelena Ginzburg, MD, Eliezer A. Rachmilewitz, MD, Patricia Giardina, MD, Robert W Grady, Stefano Rivella, PhD

Blood (2010) 116 (21): 1009.

https://doi.org/10.1182/blood.V116.21.1009.1009

Published: 2010

Journal Articles

Tmprss6 Is a Genetic Modifier of the Hfe -Hemochromatosis Phenotype in Mice.

Karin E. Finberg, MD, PhD, Rebecca Whittlesey, B.S., Mark D. Fleming, MD, DPhil, Nancy C. Andrews, MD, PhD

Blood (2009) 114 (22): 625.

https://doi.org/10.1182/blood.V114.22.625.625

Published: 2009

Journal Articles

Increased Hepcidin Expression in Mice Affected by β-Thalassemia Reduces Iron Overload with No Effect on Anemia

Sara Gardenghi, PhD, Maria Franca Marongiu, Kristen Muirhead, Pedro Ramos, Cindy N. Roy, PhD, Nancy C. Andrews, MD, PhD, Elizabeta Nemeth, PhD, Antonia Follenzi, Eliezer Rachmilewitz, M.D, Patricia Giardina, MD, Robert W Grady, PhD, Stefano Rivella, PhD

Blood (2008) 112 (11): 128.

https://doi.org/10.1182/blood.V112.11.128.128

Published: 2008

Journal Articles

Inhibited Maturation of Ter119+CD71+ Erythroid Precursors in Mice with Chronic Sterile Abscess.

Olivier D. Prince, M.D., Ian C. Prince, Jeremy Walston, MD, Nancy C. Andrews, MD, PhD, Curt I. Civin, MD, Cindy N. Roy, PhD

Blood (2008) 112 (11): 3844.

https://doi.org/10.1182/blood.V112.11.3844.3844

Published: 2008

Journal Articles

Hepcidin Expression Is Regulated by a Complex of Hemochromatosis-Associated Proteins.

Paul J. Schmidt, PhD, Franklin W. Huang, BA, Diedra M. Wrighting, BS, Paul T. Toran, BS, Nancy C. Andrews, MD, PhD

Blood (2006) 108 (11): 267.

https://doi.org/10.1182/blood.V108.11.267.267

Published: 2006

Journal Articles

Hemojuvelin Acts as a Bone Morphogenetic Protein Co-Receptor To Regulate Hepcidin Expression.

Franklin W. Huang, BS, Jodie L. Babitt, MD, Diedra M. Wrighting, MS, Tarek A. Samad, PhD, Yin Xia, PhD, Yisrael Sidis, PhD, Jason A. Campagna, Raymond T. Chung, MD, Alan L. Schneyer, PhD, Clifford J. Woolf, PhD, Nancy C. Andrews, MD, PhD, Herbert Y. Lin, MD, PhD

Blood (2005) 106 (11): 511.

https://doi.org/10.1182/blood.V106.11.511.511

Published: 2005

Journal Articles

A Mutation in Sec15l1 Disrupts the Transferrin Cycle and Causes Anemia in Hemoglobin Deficit (hbd) Mice.

Jackie E. Lim, BS, Kelly Morgan, Ph.D, Fudi Wang, MD, PhD, Mark D. Fleming, D.Phil, MD, Nancy C. Andrews, MD, PhD

Blood (2005) 106 (11): 513.

https://doi.org/10.1182/blood.V106.11.513.513

Published: 2005

Journal Articles

Iron Metabolism in the Mice with Targeted Mutations of the DMT1 Gene .

Hiromi Gunshin, PhD, Yuko Fujiwara, PhD, Angel O. Custodio, Cristina DiRenzo, Sylvie Robine, PhD, Nancy C. Andrews, MD, PhD

Blood (2004) 104 (11): 50.

https://doi.org/10.1182/blood.V104.11.50.50

Published: 2004

NARROW

Understanding the Transferrin Receptor and Cellular Iron Deficiency Outside the Erythron

Genetic Loss of Tmprss6 Increases Effective Erythropoiesis in a Mouse Model of β-Thalassemia

The Serine Protease Tmprss6 Regulates Hepcidin Expression, but Its Loss Does Not Cause Systemic Iron Deficiency In the Fetal and Neonatal Periods

Tmprss6, An Inhibitor of Hepatic Bmp/Smad Signaling, Is Required for Hepcidin Suppression and Iron Loading In a Mouse Model of β-Thalassemia

Hepcidin as a Therapeutic Tool to Limit Iron Overload and Improve Anemia In β-Thalassemia

Tmprss6 Is a Genetic Modifier of the Hfe -Hemochromatosis Phenotype in Mice.

Increased Hepcidin Expression in Mice Affected by β-Thalassemia Reduces Iron Overload with No Effect on Anemia

Inhibited Maturation of Ter119+CD71+ Erythroid Precursors in Mice with Chronic Sterile Abscess.

Hepcidin Expression Is Regulated by a Complex of Hemochromatosis-Associated Proteins.

Hemojuvelin Acts as a Bone Morphogenetic Protein Co-Receptor To Regulate Hepcidin Expression.

A Mutation in Sec15l1 Disrupts the Transferrin Cycle and Causes Anemia in Hemoglobin Deficit (hbd) Mice.

Iron Metabolism in the Mice with Targeted Mutations of the DMT1 Gene .

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American Society of Hematology

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American Society of Hematology

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