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Journal Articles

Activation of Notch-Hes Signaling Pathway Impairs Osteogenic Differentiation and Hematopoietic Stem Cell-Supporting Capacitiy of Mesenchymal Stromal Cells Derived from Myelodysplastic Syndromes Patients

Chunkang Chang, MDPhD, Chengming Fei, MD, Juan Guo, MD, Youshan Zhao, MD, Shucheng Gu, MD, Xiao Li, MD PhD

Blood (2015) 126 (23): 2857.

https://doi.org/10.1182/blood.V126.23.2857.2857

Published: 2015

Journal Articles

Labile Plasma Iron (LPI), More Practical and More Sensitive to Iron Overload in Myelodysplastic Syndromes

Chunkang Chang, MDPhD, Shucheng Gu, MD, Juan Guo, MD, Youshan Zhao, MD, Chengming Fei, MD, Xiao Li, MD PhD

Blood (2015) 126 (23): 5230.

https://doi.org/10.1182/blood.V126.23.5230.5230

Published: 2015

Journal Articles

Chidamide, a Novel Histone Deacetylase Inhibitor, Displays Potent Antitumor Activity Against MDS Cells Mainly through JAK2/STAT3 Signaling Inhibition

Chunkang Chang, MDPhD, Sida Zhao, MD, Juan Guo, MD, Youshan Zhao, MD, Chengming Fei, MD, Shucheng Gu, MD, Xiao Li, MD PhD

Blood (2015) 126 (23): 5233.

https://doi.org/10.1182/blood.V126.23.5233.5233

Published: 2015

Journal Articles

Downregulation of Dicer1 Promotes Cellular Senescence and Decreases the Differentiation and Stem Cell-Supporting Capacities of MSCs in Patients with Myelodysplastic Syndrome

Youshan Zhao, Dong Wu, Chengming Fei, Juan Guo, Shucheng Gu, Feng Xu, Zheng Zhang, Chunkang Chang, Xiao Li, MD PhD

Blood (2014) 124 (21): 4365.

https://doi.org/10.1182/blood.V124.21.4365.4365

Published: 2014

Journal Articles

Reduced Response to Stromal Cell-Derived Factor-1(SDF-1) May Lead to Low Expression of p57kip2 in the Bone Marrow of Patients with De Novo Myelodysplastic Syndromes (MDS)

Youshan Zhao, Chunkang Chang, Juan Guo, Shucheng Gu

Blood (2011) 118 (21): 2782.

https://doi.org/10.1182/blood.V118.21.2782.2782

Published: 2011

NARROW

Activation of Notch-Hes Signaling Pathway Impairs Osteogenic Differentiation and Hematopoietic Stem Cell-Supporting Capacitiy of Mesenchymal Stromal Cells Derived from Myelodysplastic Syndromes Patients

Labile Plasma Iron (LPI), More Practical and More Sensitive to Iron Overload in Myelodysplastic Syndromes

Chidamide, a Novel Histone Deacetylase Inhibitor, Displays Potent Antitumor Activity Against MDS Cells Mainly through JAK2/STAT3 Signaling Inhibition

Downregulation of Dicer1 Promotes Cellular Senescence and Decreases the Differentiation and Stem Cell-Supporting Capacities of MSCs in Patients with Myelodysplastic Syndrome

Reduced Response to Stromal Cell-Derived Factor-1(SDF-1) May Lead to Low Expression of p57kip2 in the Bone Marrow of Patients with De Novo Myelodysplastic Syndromes (MDS)

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ASH Publications

American Society of Hematology

Activation of Notch-Hes Signaling Pathway Impairs Osteogenic Differentiation and Hematopoietic Stem Cell-Supporting Capacitiy of Mesenchymal Stromal Cells Derived from Myelodysplastic Syndromes Patients

Labile Plasma Iron (LPI), More Practical and More Sensitive to Iron Overload in Myelodysplastic Syndromes

Chidamide, a Novel Histone Deacetylase Inhibitor, Displays Potent Antitumor Activity Against MDS Cells Mainly through JAK2/STAT3 Signaling Inhibition

Downregulation of Dicer1 Promotes Cellular Senescence and Decreases the Differentiation and Stem Cell-Supporting Capacities of MSCs in Patients with Myelodysplastic Syndrome

Reduced Response to Stromal Cell-Derived Factor-1(SDF-1) May Lead to Low Expression of p57kip2 in the Bone Marrow of Patients with De Novo Myelodysplastic Syndromes (MDS)

Email alerts

ASH Publications

American Society of Hematology

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