Page 1 | Search Results | Blood | American Society of Hematology

1-9 of 9

Sort by

Sort Order Select

Journal Articles

NFIB: A Gene Consistently Upregulated in MPD and Frequently Co-Amplified with JAK2V617F Augments Growth of Erythropoietin Dependent Cells

Xiaoqing Lin, BS, Monica Buzzai, PhD, Ross L Levine, MD, Windy Berkofsky-Fessler, PhD, Weijia Zhang, PhD, Benjamin L. Ebert, MD, DPhil, D. Gary Gilliland, MD, PhD, Jonathan D. Licht, MD

Blood (2008) 112 (11): 3727.

https://doi.org/10.1182/blood.V112.11.3727.3727

Published: 2008

Journal Articles

Integrative Analysis of Genomic Aberration and Gene Expression Suggests That MPD with Loss of Heterozygosity or Gain of Chromosome 9 Represents a Distinct Entity

Weijia Zhang, PhD, Benjamin L. Ebert, MD, DPhil, Windy Berkofsky-Fessler, PhD, Monica Buzzai, PhD, Yezhou Sun, MSc, Caixia Xi, MSc, Ross L Levine, MD, D. Gary Gilliland, MD, PhD, Jonathan D. Licht, MD

Blood (2008) 112 (11): 3735.

https://doi.org/10.1182/blood.V112.11.3735.3735

Published: 2008

Journal Articles

Myelodysplastic Syndrome (MDS) Displays Profound and Functionally Significant Epigenetic Deregulation Compared to Acute Myeloid Leukemia (AML) and Normal Bone Marrow Cells.

Maria E. Figueroa, MD, Tamer Fandy, MSC PhD, Melanie J. McConnell, PhD, Windy Berkofsky-Fessler, PhD, Chris Nasrallah, MS, Hetty Carraway, MD, Jonathan Licht, MD, Steven D. Gore, MD, Ari M. Melnick, MD

Blood (2007) 110 (11): 345.

https://doi.org/10.1182/blood.V110.11.345.345

Published: 2007

Journal Articles

A Comprehensive Genomic Approach Using Gain of Function and Loss of Function Cell Models and ChIP-on-Chip Technology Identifies Novel Promyelocytic Zinc Finger Protein Target Genes.

Kim L. Rice, PhD, Ari Melnick, MD Ph.D, Kenny Ye, Ph.D, Windy Berkofsky-Fessler, Jonathan D. Licht, MD

Blood (2006) 108 (11): 1407.

https://doi.org/10.1182/blood.V108.11.1407.1407

Published: 2006

Journal Articles

Engineered Expression of JAK2V617F Drastically Changes the Response of Progenitor Cells to Erythropoietin and Partially Mimics the Polycythemia Expression Signature.

Windy Berkofsky-Fessler, MPhil, Melanie-Jane McConnell, PhD, Donna S. Neuberg, DSc, Jonathan D. Licht, MD

Blood (2006) 108 (11): 3597.

https://doi.org/10.1182/blood.V108.11.3597.3597

Published: 2006

Journal Articles

High Resolution SNP Arrays and Correlation with Expression Arrays Identify Novel Genomic Aberrations and Potential Disease Genes in Polycythemia Vera and Other Myeloproliferative Disorders.

Weijia Zhang, PhD, Windy Berkofsky-Fessler, MPhil, Ross Levine, MD, Yezhou Sun, MS, D. Gary Gilliland, MD, Jonathan D. Licht, MD

Blood (2006) 108 (11): 359.

https://doi.org/10.1182/blood.V108.11.359.359

Published: 2006

Journal Articles

FISH Studies Provide Evidence That JAK 2 , Via a Myriad of Mechanisms Not Limited to Point Mutation, Has Gain of Function in Polycythemia Vera (PV), and Myelodysplasia (MDS).

Vesna Najfeld, PhD, Windy Berkofsky-Fessler, M.Sc., Amanda Cozza, B.Sc

Blood (2006) 108 (11): 2697.

https://doi.org/10.1182/blood.V108.11.2697.2697

Published: 2006

Journal Articles

A Phase I-II Pilot Study of Divalproex Sodium Alone and in Combination with All-Trans-Retinoic Acid (ATRA) in Relapsed or Refractory Acute Myeloid Leukemia.

Janice L. Gabrilove, MD, Melanie-Jane McConnell, MD, Windy Berkofsky-Fessler, MD, Andres L. Sirulnik, MD, PhD, Sanjay Sharma, MD, Lewis R. Silverman, MD, Luis Isola, MD, John Mandeli, PhD, Jonathan Licht, MD

Blood (2005) 106 (11): 2799.

https://doi.org/10.1182/blood.V106.11.2799.2799

Published: 2005

Journal Articles

The Transcriptional Profile of PV Displays Limited Similarity to EPO Stimulated Progenitor Cells: Evidence That JAK2 V617F Confers a Novel Program to Malignant Hematopoietic Stem Cells.

Windy Berkofsky-Fessler, MPhil, Melanie J. McConnell, PhD, Kai Huang, Monica L. Bailey, Dwayne L. Barber, PhD, Ross L. Levine, MD, Donna S. Neuberg, DSc, D. Gary Gilliland, MD, PhD, Jonathan D. Licht, MD

Blood (2005) 106 (11): 120.

https://doi.org/10.1182/blood.V106.11.120.120

Published: 2005

NARROW

NFIB: A Gene Consistently Upregulated in MPD and Frequently Co-Amplified with JAK2V617F Augments Growth of Erythropoietin Dependent Cells

Integrative Analysis of Genomic Aberration and Gene Expression Suggests That MPD with Loss of Heterozygosity or Gain of Chromosome 9 Represents a Distinct Entity

Myelodysplastic Syndrome (MDS) Displays Profound and Functionally Significant Epigenetic Deregulation Compared to Acute Myeloid Leukemia (AML) and Normal Bone Marrow Cells.

A Comprehensive Genomic Approach Using Gain of Function and Loss of Function Cell Models and ChIP-on-Chip Technology Identifies Novel Promyelocytic Zinc Finger Protein Target Genes.

Engineered Expression of JAK2V617F Drastically Changes the Response of Progenitor Cells to Erythropoietin and Partially Mimics the Polycythemia Expression Signature.

High Resolution SNP Arrays and Correlation with Expression Arrays Identify Novel Genomic Aberrations and Potential Disease Genes in Polycythemia Vera and Other Myeloproliferative Disorders.

FISH Studies Provide Evidence That JAK 2 , Via a Myriad of Mechanisms Not Limited to Point Mutation, Has Gain of Function in Polycythemia Vera (PV), and Myelodysplasia (MDS).

A Phase I-II Pilot Study of Divalproex Sodium Alone and in Combination with All-Trans-Retinoic Acid (ATRA) in Relapsed or Refractory Acute Myeloid Leukemia.

The Transcriptional Profile of PV Displays Limited Similarity to EPO Stimulated Progenitor Cells: Evidence That JAK2 V617F Confers a Novel Program to Malignant Hematopoietic Stem Cells.

Email alerts

ASH Publications

American Society of Hematology

Email alerts

ASH Publications

American Society of Hematology

This Feature Is Available To Subscribers Only