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Journal Articles

Vitamin D Repletion Mitigates Oxidative Stress Induced Pulmonary Artery Remodeling in Sickle Cell Disease

Julia Brittain, PhD, E. Leila Jerome Clay, MD, Ryan Harris, PhD CEP, FACSM, Itia Lee, BS, Abdullah Kutlar, MD, Sanjiv Kumar, PhD

Blood (2017) 130 (Supplement 1): 443.

https://doi.org/10.1182/blood.V130.Suppl_1.443.443

Published: 2017

Journal Articles

Thiocyanate Blocks Peroxidase-Dependent Extracellular Trap (ET) Formation By PMN and Eosinophils: Heme Is a Potent New Agonist For The ET Pathway

Kristin Mathson, B.S., Rehani Teena, Ph.D., John D Belcher, Ph.D., Gregory M Vercellotti, Arne Slungaard, MD

Blood (2013) 122 (21): 323.

https://doi.org/10.1182/blood.V122.21.323.323

Published: 2013

Journal Articles

Expression of the secondary granule proteins major basic protein 1 (MBP-1) and eosinophil peroxidase (EPX) is required for eosinophilopoiesis in mice

Alfred D. Doyle, Elizabeth A. Jacobsen, Sergei I. Ochkur, Michael P. McGarry, Kevin G. Shim, David T. C. Nguyen, Cheryl Protheroe, Dana Colbert, Jake Kloeber, Joseph Neely, Kelly P. Shim, Kimberly D. Dyer, Helene F. Rosenberg, James J. Lee, Nancy A. Lee

Blood (2013) 122 (5): 781–790.

https://doi.org/10.1182/blood-2013-01-473405

Published: 2013

Includes: Supplemental data

Journal Articles

Increased Mutation Frequency Induced by Oxidative Stress in the NUP98-HOXD13 MDS Mouse Model

Yang Jo Chung, PhD, Carine Robert, PhD, Sheryl M Gough, BSc, PhD, Feyruz Rassool, PhD, Peter Aplan, MD

Blood (2011) 118 (21): 1705.

https://doi.org/10.1182/blood.V118.21.1705.1705

Published: 2011

Journal Articles

The Major Phagocyte Peroxidase Product HOSCN Stimulates PMN and Eosinophil Adhesion to Endothelium by p38 MAPK-Dependent Enhancement of CD11b/18-ICAM-1 Binding,

Adria Jonas, M.S., Teena Rehani, Ph.D., Arne Slungaard, MD

Blood (2011) 118 (21): 3205.

https://doi.org/10.1182/blood.V118.21.3205.3205

Published: 2011

Journal Articles

The Major Phagocyte Peroxidase-Derived Oxidant HOSCN Stimulates Gene-Specific p38 MAPK- Dependent, NF-κb-Mediated Activation of Tissue Factor, VCAM-1 and ICAM-1 Expression In Endothelium.

Teena Rehani, Ph.D., Adria Jonas, M.S., Arne Slungaard, MD

Blood (2010) 116 (21): 1481.

https://doi.org/10.1182/blood.V116.21.1481.1481

Published: 2010

Journal Articles

Differentiation of Mature Eosinophils From Human Embryonic and Induced Pluripotent Stem Cells.

Wenyu Yang, Feng Ma, MD, PhD, Kenji Matsumoto, Natsumi Nishihama, Hiroshi Sagara, Yasuhiro Ebihara, MD, PhD, Yanzheng Gu, Hirohisa Saito, MD, PhD, Xiaofan Zhu, MD, PhD, Hiromitsu Nakauchi, MD, PhD, Kohichiro Tsuji, MD, PhD

Blood (2010) 116 (21): 1594.

https://doi.org/10.1182/blood.V116.21.1594.1594

Published: 2010

Journal Articles

Altered Functional Properties of Eosinophils In Sickle Cell Anemia and Effects of Hydroxyurea Therapy

Flavia Rubia Pallis, B.Sc., Nicola Conran, PhD, Kleber Yotsumoto Fertrin, MD, Sara T. Olalla-Saad, M.D., Ph.D., Fernando Ferreira Costa, Ph.D., M.D., Carla Fernanda Franco-Penteado, PhD

Blood (2010) 116 (21): 2656.

https://doi.org/10.1182/blood.V116.21.2656.2656

Published: 2010

Journal Articles

Gene-Specific Effects of the Histone Deacetylase Inhibitor TSA on Activation of a Pro-Inflammatory Endothelial Cell Phenotype by the Major Phagocyte Peroxidase-Derived Oxidant HOSCN.

Teena Pasrija, Ph.D., Shawn A Mahmud, Ph.D., John D Belcher, Ph.D., Gregory M. Vercellotti, MD, Arne Slungaard, MD

Blood (2008) 112 (11): 1274.

https://doi.org/10.1182/blood.V112.11.1274.1274

Published: 2008

Journal Articles

Histopathology of experimentally induced asthma in a murine model of sickle cell disease

Sandhya D. Nandedkar, Thomas R. Feroah, William Hutchins, Dorothee Weihrauch, Kameswari S. Konduri, Jingli Wang, Robert C. Strunk, Michael R. DeBaun, Cheryl A. Hillery, Kirkwood A. Pritchard

Blood (2008) 112 (6): 2529–2538.

https://doi.org/10.1182/blood-2008-01-132506

Published: 2008

Includes: Supplemental data

Journal Articles

The Major Phagocyte Peroxidase-Derived Oxidant, HOSCN (Hypothiocyanous Acid), Induces Proinflammatory and Cytoprotective Gene Expression in Endothelium: A Mechanism for Microlocalized Regulation of Inflammation.

Shawn A. Mahmud, BA, Jian-Guo Wang, Ph.D., Peng Wei, M.S., Molly S. Thomas, Ph.D., John D. Belcher, Ph.D., Gregory M. Vercellotti, M.D., Arne Slungaard, M.D.

Blood (2007) 110 (11): 3295.

https://doi.org/10.1182/blood.V110.11.3295.3295

Published: 2007

Journal Articles

Diverse Myeloid Lineage Potential Arises in the Yolk Sac of the Mammalian Embryo.

James Palis, MD, Anne Koniski, MS, Timothy P. Bushnell, PhD, Kathleen E. McGrath, PhD

Blood (2006) 108 (11): 1666.

https://doi.org/10.1182/blood.V108.11.1666.1666

Published: 2006

Journal Articles

Thiocyanate-Dependent Inhibition of Spontaneous and Agonist-Induced Eosinophil Apoptosis by Eosinophil Peroxidase (EPO): A Potential Physiologic Role for Endogenously Generated HOSCN in Maintaining Eosinophil Viability.

Shawn A. Mahmud, BS, Jianguo Wang, Ph.D., Arne Slungaard, M.D.

Blood (2006) 108 (11): 1633.

https://doi.org/10.1182/blood.V108.11.1633.1633

Published: 2006

Journal Articles

The principal eosinophil peroxidase product, HOSCN, is a uniquely potent phagocyte oxidant inducer of endothelial cell tissue factor activity: a potential mechanism for thrombosis in eosinophilic inflammatory states

Jian-Guo Wang, Shawn A. Mahmud, Jacob A. Thompson, Jian-Guo Geng, Nigel S. Key, Arne Slungaard

Blood (2006) 107 (2): 558–565.

https://doi.org/10.1182/blood-2005-05-2152

Published: 2006

Journal Articles

Developmental and Differential Changes of Gene Expression in Erythroid Progenitor Cells.

Vladan P. Cokic, MD, Bhaskar Bhattacharya, PhD, Raj K. Puri, MD, Alan N. Schechter, MD

Blood (2005) 106 (11): 3648.

https://doi.org/10.1182/blood.V106.11.3648.3648

Published: 2005

Journal Articles

The Principal Eosinophil Peroxidase Oxidant, HOSCN, Is a Uniquely Potent Phagocyte Oxidant Inducer of E-Selectin, ICAM-1 and VCAM-1 Expression on Endothelial Cells and Promotes Leukocyte-Endothelial Cell Adhesion. A Potential Mechanism Regulating Leukocyte Trafficking in Eosinophilic Inflammatory States.

Jianguo Wang, PhD, Rong Tang, PhD, Yuqing Huo, MD, PhD, Arne Slungaard, M.D

Blood (2005) 106 (11): 3085.

https://doi.org/10.1182/blood.V106.11.3085.3085

Published: 2005

Journal Articles

The Principal Eosinophil Peroxidase Oxidant, HOSCN, Potently Induces Endothelial Cell Tissue Factor Activity through Transcriptional Activation Dependent upon NF-kB and ERK1/2: Potential Mechanism for a Prothrombotic and Proinflammatory Endothelial Phenotype in the Hypereosinophilic Syndrome.

Jianguo Wang, Ph.D., Shawn A. Mahmud, B.S., Jake A. Thompson, B.S., Jianguo Geng, M.D./Ph.D., Nigel S. Key, M.D., Arne Slungaard, M.D.

Blood (2004) 104 (11): 804.

https://doi.org/10.1182/blood.V104.11.804.804

Published: 2004

Journal Articles

Regulation of neutrophil and eosinophil secondary granule gene expression by transcription factors C/EBPε and PU.1

Adrian F. Gombart, Scott H. Kwok, Karen L. Anderson, Yuji Yamaguchi, Bruce E. Torbett, H. Phillip Koeffler

Blood (2003) 101 (8): 3265–3273.

https://doi.org/10.1182/blood-2002-04-1039

Published: 2003

NARROW

Vitamin D Repletion Mitigates Oxidative Stress Induced Pulmonary Artery Remodeling in Sickle Cell Disease

Thiocyanate Blocks Peroxidase-Dependent Extracellular Trap (ET) Formation By PMN and Eosinophils: Heme Is a Potent New Agonist For The ET Pathway

Expression of the secondary granule proteins major basic protein 1 (MBP-1) and eosinophil peroxidase (EPX) is required for eosinophilopoiesis in mice

Increased Mutation Frequency Induced by Oxidative Stress in the NUP98-HOXD13 MDS Mouse Model

The Major Phagocyte Peroxidase Product HOSCN Stimulates PMN and Eosinophil Adhesion to Endothelium by p38 MAPK-Dependent Enhancement of CD11b/18-ICAM-1 Binding,

The Major Phagocyte Peroxidase-Derived Oxidant HOSCN Stimulates Gene-Specific p38 MAPK- Dependent, NF-κb-Mediated Activation of Tissue Factor, VCAM-1 and ICAM-1 Expression In Endothelium.

Differentiation of Mature Eosinophils From Human Embryonic and Induced Pluripotent Stem Cells.

Altered Functional Properties of Eosinophils In Sickle Cell Anemia and Effects of Hydroxyurea Therapy

Gene-Specific Effects of the Histone Deacetylase Inhibitor TSA on Activation of a Pro-Inflammatory Endothelial Cell Phenotype by the Major Phagocyte Peroxidase-Derived Oxidant HOSCN.

Histopathology of experimentally induced asthma in a murine model of sickle cell disease

The Major Phagocyte Peroxidase-Derived Oxidant, HOSCN (Hypothiocyanous Acid), Induces Proinflammatory and Cytoprotective Gene Expression in Endothelium: A Mechanism for Microlocalized Regulation of Inflammation.

Diverse Myeloid Lineage Potential Arises in the Yolk Sac of the Mammalian Embryo.

Thiocyanate-Dependent Inhibition of Spontaneous and Agonist-Induced Eosinophil Apoptosis by Eosinophil Peroxidase (EPO): A Potential Physiologic Role for Endogenously Generated HOSCN in Maintaining Eosinophil Viability.

The principal eosinophil peroxidase product, HOSCN, is a uniquely potent phagocyte oxidant inducer of endothelial cell tissue factor activity: a potential mechanism for thrombosis in eosinophilic inflammatory states

Developmental and Differential Changes of Gene Expression in Erythroid Progenitor Cells.

The Principal Eosinophil Peroxidase Oxidant, HOSCN, Potently Induces Endothelial Cell Tissue Factor Activity through Transcriptional Activation Dependent upon NF-kB and ERK1/2: Potential Mechanism for a Prothrombotic and Proinflammatory Endothelial Phenotype in the Hypereosinophilic Syndrome.

Regulation of neutrophil and eosinophil secondary granule gene expression by transcription factors C/EBPε and PU.1

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American Society of Hematology

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American Society of Hematology

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