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Journal Articles

The Tumor Suppressor APC/C Cdh1 and Its Role In Replication Stress and The Origin Of Genomic Instability

Julika Krohs, Dominik Schnerch, MD, Marie Follo, Julia Felthaus, Monika Engelhardt, MD, Ralph M. Waesch, MD

Blood (2013) 122 (21): 2489.

https://doi.org/10.1182/blood.V122.21.2489.2489

Published: 2013

Journal Articles

APC/C Cdh1 As a Mediator Of Hematopoietic Differentiation, Self-Renewal and Malignant Transformation

Andrea Schmidts, Daniel Ewerth, Dominik Schnerch, MD, Arunas Kvainickas, Dagmar Wider, Birgit Kügelgen, Heike L. Pahl, PhD, Monika Engelhardt, MD, Ralph Wäsch, MD

Blood (2013) 122 (21): 3676.

https://doi.org/10.1182/blood.V122.21.3676.3676

Published: 2013

Journal Articles

A Rationale to Enhance the Response to Antimitotic Therapy in Acute Myeloid Leukemia

Dominik Schnerch, MD, Julia Felthaus, Monika Engelhardt, Ralph Wäsch, MD

Blood (2012) 120 (21): 1332.

https://doi.org/10.1182/blood.V120.21.1332.1332

Published: 2012

Journal Articles

Multikinase Inhibitor Sorafenib Induces Apoptosis, CD138-Downregulation, Actin Depolymerization and Inhibition of M210B4-Triggered Chemotaxis in Multiple Myeloma Cell Lines and Synergyzes with Proteasome Inhibitor Bortezomib

Josefina Udi, MD, Dagmar Wider, Julie Catusse, PhD, Dominik Schnerch, MD, Marie Follo, PhD, Gabriele Ihorst, Ralph Waesch, MD, Monika Engelhardt, MD

Blood (2011) 118 (21): 2380.

https://doi.org/10.1182/blood.V118.21.2380.2380

Published: 2011

Journal Articles

The Spindle Checkpoint Protein BubR1 Is Differentially Expressed in the Lymphatic and Myeloid Lineage and Is a Determinator of Response to Spindle Poisons

Dominik Schnerch, MD, Marie Follo, PhD, Andrea Schmidts, Julika Krohs, Julia Felthaus, Monika Engelhardt, MD, Ralph Wäsch

Blood (2011) 118 (21): 1407.

https://doi.org/10.1182/blood.V118.21.1407.1407

Published: 2011

NARROW

The Tumor Suppressor APC/C Cdh1 and Its Role In Replication Stress and The Origin Of Genomic Instability

APC/C Cdh1 As a Mediator Of Hematopoietic Differentiation, Self-Renewal and Malignant Transformation

A Rationale to Enhance the Response to Antimitotic Therapy in Acute Myeloid Leukemia

Multikinase Inhibitor Sorafenib Induces Apoptosis, CD138-Downregulation, Actin Depolymerization and Inhibition of M210B4-Triggered Chemotaxis in Multiple Myeloma Cell Lines and Synergyzes with Proteasome Inhibitor Bortezomib

The Spindle Checkpoint Protein BubR1 Is Differentially Expressed in the Lymphatic and Myeloid Lineage and Is a Determinator of Response to Spindle Poisons

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ASH Publications

American Society of Hematology

The Tumor Suppressor APC/C Cdh1 and Its Role In Replication Stress and The Origin Of Genomic Instability

APC/C Cdh1 As a Mediator Of Hematopoietic Differentiation, Self-Renewal and Malignant Transformation

A Rationale to Enhance the Response to Antimitotic Therapy in Acute Myeloid Leukemia

Multikinase Inhibitor Sorafenib Induces Apoptosis, CD138-Downregulation, Actin Depolymerization and Inhibition of M210B4-Triggered Chemotaxis in Multiple Myeloma Cell Lines and Synergyzes with Proteasome Inhibitor Bortezomib

The Spindle Checkpoint Protein BubR1 Is Differentially Expressed in the Lymphatic and Myeloid Lineage and Is a Determinator of Response to Spindle Poisons

Email alerts

ASH Publications

American Society of Hematology

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