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A new severe congenital neutropenia syndrome associated with autosomal recessive COPZ1 mutations

Blood (2025) 145 (20): 2317–2335.

https://doi.org/10.1182/blood.2023022576

Published: 2025

Includes: Supplemental data

Journal Articles

Autosomal Recessive COPZ1 Mutations Cause Isolated Severe Neutropenia or Complex Syndrome with Severe Neutropenia

Blood (2024) 144 (Supplement 1): 417.

https://doi.org/10.1182/blood-2024-199069

Published: 2024

Journal Articles

The Clinical-Stage sGC Stimulator IW-1701 Prevents Increase of Plasma Biomarkers of Intravascular Inflammation and Suppresses Leukocyte-Endothelial Interactions in Tnfα-Treated Mice

Boris Tchernychev, Susanne Feil, Peter Germano, William Warren, Elisabeth Lonie, Robert Feil, Todd G Milne, John Hadcock, Yueh-Tyng Chien, Mark G Currie, Regina Margaret Graul

Blood (2017) 130 (Supplement 1): 447.

https://doi.org/10.1182/blood.V130.Suppl_1.447.447

Published: 2017

Journal Articles

Sequential activation of p38 and ERK pathways by cGMP-dependent protein kinase leading to activation of the platelet integrin α IIb β 3

Zhenyu Li, Guoying Zhang, Robert Feil, Jiahuai Han, Xiaoping Du

Blood (2006) 107 (3): 965–972.

https://doi.org/10.1182/blood-2005-03-1308

Published: 2006

Journal Articles

The Role of the cGMP-Dependent Protein Kinase II in Platelet Activation and In Vivo Thrombosis.

Zhenyu Li, MD, PhD, Guoying Zhang, MD, Hong Yin, MD, Robert Feil, PhD, Franz Hofmann, PhD, Xiaoping Du, MD, PhD

Blood (2005) 106 (11): 653.

https://doi.org/10.1182/blood.V106.11.653.653

Published: 2005

Journal Articles

GPIb-dependent platelet activation is dependent on Src kinases but not MAP kinase or cGMP-dependent kinase

Stuart J. Marshall, Yotis A. Senis, Jocelyn M. Auger, Robert Feil, Franz Hofmann, Gary Salmon, J. Thomas Peterson, Frank Burslem, Steve P. Watson

Blood (2004) 103 (7): 2601–2609.

https://doi.org/10.1182/blood-2003-09-3319

Published: 2004

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A new severe congenital neutropenia syndrome associated with autosomal recessive COPZ1 mutations

Autosomal Recessive COPZ1 Mutations Cause Isolated Severe Neutropenia or Complex Syndrome with Severe Neutropenia

The Clinical-Stage sGC Stimulator IW-1701 Prevents Increase of Plasma Biomarkers of Intravascular Inflammation and Suppresses Leukocyte-Endothelial Interactions in Tnfα-Treated Mice

Sequential activation of p38 and ERK pathways by cGMP-dependent protein kinase leading to activation of the platelet integrin α IIb β 3

The Role of the cGMP-Dependent Protein Kinase II in Platelet Activation and In Vivo Thrombosis.

GPIb-dependent platelet activation is dependent on Src kinases but not MAP kinase or cGMP-dependent kinase

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ASH Publications

American Society of Hematology

Update search

A new severe congenital neutropenia syndrome associated with autosomal recessive COPZ1 mutations

Autosomal Recessive COPZ1 Mutations Cause Isolated Severe Neutropenia or Complex Syndrome with Severe Neutropenia

The Clinical-Stage sGC Stimulator IW-1701 Prevents Increase of Plasma Biomarkers of Intravascular Inflammation and Suppresses Leukocyte-Endothelial Interactions in Tnfα-Treated Mice

Sequential activation of p38 and ERK pathways by cGMP-dependent protein kinase leading to activation of the platelet integrin α IIb β 3

The Role of the cGMP-Dependent Protein Kinase II in Platelet Activation and In Vivo Thrombosis.

GPIb-dependent platelet activation is dependent on Src kinases but not MAP kinase or cGMP-dependent kinase

Email alerts

ASH Publications

American Society of Hematology

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