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Journal Articles

Flippases and Scramblases at Plasma Membranes that Regulate Phosphatidylserine Exposure

Shigekazu Nagata, PhD

Blood (2015) 126 (23): SCI-31.

https://doi.org/10.1182/blood.V126.23.SCI-31.SCI-31

Published: 2015

Journal Articles

Platelet Phosphatidylserine Exposure, Survival and Blood Coagulation in Mice Lacking TMEM16F

Gary E. Gilbert, MD, Colin Kretz, PhD, David Ginsburg, MD, Valerie Anne Novakovic, MS

Blood (2015) 126 (23): SCI-32.

https://doi.org/10.1182/blood.V126.23.SCI-32.SCI-32

Published: 2015

Journal Articles

Regulation of Platelet Procoagulant Activity

Johan Heemskerk, PhD

Blood (2015) 126 (23): SCI-33.

https://doi.org/10.1182/blood.V126.23.SCI-33.SCI-33

Published: 2015

Journal Articles

TMEM16F Deficiency Causes a Strain-Dependent Lethality in Mice and Reduced Phosphatidylserine Exposure on Heterozygous As Well As Homozygous Deficient Mouse Platelets

Colin A Kretz, PhD, Valerie A Novakovic, M.S., Anna Kiseleva, Audrey C.A. Cleuren, PhD, Rami Khoriaty, MD, Elizabeth Adams, PhD, David Ginsburg, MD, Gary E. Gilbert, MD

Blood (2014) 124 (21): 4157.

https://doi.org/10.1182/blood.V124.21.4157.4157

Published: 2014

Journal Articles

Both TMEM16F-dependent and TMEM16F-independent pathways contribute to phosphatidylserine exposure in platelet apoptosis and platelet activation

Roger van Kruchten, Nadine J. A. Mattheij, Christine Saunders, Marion A. H. Feijge, Frauke Swieringa, Jef L. N. Wolfs, Peter W. Collins, Johan W. M. Heemskerk, Edouard M. Bevers

Blood (2013) 121 (10): 1850–1857.

https://doi.org/10.1182/blood-2012-09-454314

Published: 2013

Includes: Supplemental data

Journal Articles

Putative Phospholipid “scramblase” of Scott Syndrome, TMEM16F/Ano6, Mediates Phosphatidylserine Exposure On Filopodia and Cell Margins of Viable Endothelial Cells.

Jialan Shi, MD, PhD, Gary E. Gilbert, M.D.

Blood (2012) 120 (21): 2180.

https://doi.org/10.1182/blood.V120.21.2180.2180

Published: 2012

Journal Articles

TMEM16F Ion Channel Regulates Calcium-Dependent PS Exposure, Hemostasis, and Thrombosis

Andrew Y. Kim, PhD, Huanghe Yang, PhD, Tovo David, PhD, Jason Tien, Shaun R Coughlin, PhD, Yuh Nung Jan, Ph.D., Lily Jan, PhD

Blood (2012) 120 (21): 1111.

https://doi.org/10.1182/blood.V120.21.1111.1111

Published: 2012

Journal Articles

Compound heterozygosity for 2 novel TMEM16F mutations in a patient with Scott syndrome

Elisabetta Castoldi, Peter W. Collins, Patrick L. Williamson, Edouard M. Bevers

Blood (2011) 117 (16): 4399–4400.

https://doi.org/10.1182/blood-2011-01-332502

Published: 2011

Journal Articles

Scott Syndrome Dogs Demonstrate a Failure of Coated-Platelet Formation.

Marjory B. Brooks, DVM, James L. Catalfamo, PhD, Paul Friese, BS, George L. Dale, PhD

Blood (2006) 108 (11): 1104.

https://doi.org/10.1182/blood.V108.11.1104.1104

Published: 2006

Journal Articles

Platelet ABCA1 Expression Is Normal in Scott Syndrome Dogs.

James L. Catalfamo, PhD, Marjory B. Brooks, DVM, Melanie Ammersbach, BS, Amanda K. DeMaster, BS

Blood (2006) 108 (11): 3918.

https://doi.org/10.1182/blood.V108.11.3918.3918

Published: 2006

Journal Articles

Reversible inhibition of the platelet procoagulant response through manipulation of the Gardos channel

Jef L. Wolfs, Simone J. Wielders, Paul Comfurius, Theo Lindhout, John C. Giddings, Robert F. Zwaal, Edouard M. Bevers

Blood (2006) 108 (7): 2223–2228.

https://doi.org/10.1182/blood-2006-01-009613

Published: 2006

Journal Articles

An ABC for Scott syndrome?

Florence Toti, Jean-Marie Freyssinet

Blood (2005) 106 (2): 396–397.

https://doi.org/10.1182/blood-2005-04-1716

Published: 2005

Journal Articles

A novel missense mutation in ABCA1 results in altered protein trafficking and reduced phosphatidylserine translocation in a patient with Scott syndrome

Christiane Albrecht, John H. McVey, James I. Elliott, Alessandro Sardini, Ildiko Kasza, Andrew D. Mumford, Rossi P. Naoumova, Edward G. D. Tuddenham, Katalin Szabo, Christopher F. Higgins

Blood (2005) 106 (2): 542–549.

https://doi.org/10.1182/blood-2004-05-2056

Published: 2005

Journal Articles

Characterization of a Heritable Bleeding Disorder in a Horse: Reduce PS-Flip Leads to Progressive Dysfunction in the Common Pathway of Platelet Coagulation.

Jeffrey W. Norris, PhD, Joong-Hyuck Auh, PhD, Sandra Wilson, BS, Dana S. Clutter, BS, John H. Crowe, PhD, Fern Tablin, VMD, PhD

Blood (2004) 104 (11): 3932.

https://doi.org/10.1182/blood.V104.11.3932.3932

Published: 2004

Journal Articles

Expression of Proteins Controlling Transbilayer Movement of Plasma Membrane Phospholipids in the B Lymphocytes From a Patient With Scott Syndrome

Quansheng Zhou, Peter J. Sims, Therese Wiedmer

Blood (1998) 92 (5): 1707–1712.

https://doi.org/10.1182/blood.V92.5.1707

Published: 1998

Journal Articles

Impaired Ca 2+ -Induced Tyrosine Phosphorylation and Defective Lipid Scrambling in Erythrocytes From a Patient With Scott Syndrome: A Study Using an Inhibitor for Scramblase That Mimics the Defect in Scott Syndrome

David W.C. Dekkers, Paul Comfurius, Wim M.J. Vuist, Jeffrey T. Billheimer, Ira Dicker, Harvey J. Weiss, Robert F. A. Zwaal, Edouard M. Bevers

Blood (1998) 91 (6): 2133–2138.

https://doi.org/10.1182/blood.V91.6.2133

Published: 1998

Journal Articles

Platelet Prothrombinase Activity and Intracellular Calcium Responses in Patients With Storage Pool Deficiency, Glycoprotein IIb-IIIa Deficiency, or Impaired Platelet Coagulant Activity — A Comparison With Scott Syndrome

Harvey J. Weiss, Bruce Lages

Blood (1997) 89 (5): 1599–1611.

https://doi.org/10.1182/blood.V89.5.1599

Published: 1997

NARROW

Flippases and Scramblases at Plasma Membranes that Regulate Phosphatidylserine Exposure

Platelet Phosphatidylserine Exposure, Survival and Blood Coagulation in Mice Lacking TMEM16F

Regulation of Platelet Procoagulant Activity

TMEM16F Deficiency Causes a Strain-Dependent Lethality in Mice and Reduced Phosphatidylserine Exposure on Heterozygous As Well As Homozygous Deficient Mouse Platelets

Both TMEM16F-dependent and TMEM16F-independent pathways contribute to phosphatidylserine exposure in platelet apoptosis and platelet activation

Putative Phospholipid “scramblase” of Scott Syndrome, TMEM16F/Ano6, Mediates Phosphatidylserine Exposure On Filopodia and Cell Margins of Viable Endothelial Cells.

TMEM16F Ion Channel Regulates Calcium-Dependent PS Exposure, Hemostasis, and Thrombosis

Compound heterozygosity for 2 novel TMEM16F mutations in a patient with Scott syndrome

Scott Syndrome Dogs Demonstrate a Failure of Coated-Platelet Formation.

Platelet ABCA1 Expression Is Normal in Scott Syndrome Dogs.

Reversible inhibition of the platelet procoagulant response through manipulation of the Gardos channel

An ABC for Scott syndrome?

A novel missense mutation in ABCA1 results in altered protein trafficking and reduced phosphatidylserine translocation in a patient with Scott syndrome

Characterization of a Heritable Bleeding Disorder in a Horse: Reduce PS-Flip Leads to Progressive Dysfunction in the Common Pathway of Platelet Coagulation.

Expression of Proteins Controlling Transbilayer Movement of Plasma Membrane Phospholipids in the B Lymphocytes From a Patient With Scott Syndrome

Impaired Ca 2+ -Induced Tyrosine Phosphorylation and Defective Lipid Scrambling in Erythrocytes From a Patient With Scott Syndrome: A Study Using an Inhibitor for Scramblase That Mimics the Defect in Scott Syndrome

Platelet Prothrombinase Activity and Intracellular Calcium Responses in Patients With Storage Pool Deficiency, Glycoprotein IIb-IIIa Deficiency, or Impaired Platelet Coagulant Activity — A Comparison With Scott Syndrome

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American Society of Hematology

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ASH Publications

American Society of Hematology

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