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Journal Articles

Mouse hematopoietic cell–targeted STAT3 deletion: stem/progenitor cell defects, mitochondrial dysfunction, ROS overproduction, and a rapid aging–like phenotype

Charlie Mantel, Steven Messina-Graham, Akira Moh, Scott Cooper, Giao Hangoc, Xin-Yuan Fu, Hal E. Broxmeyer

Blood (2012) 120 (13): 2589–2599.

https://doi.org/10.1182/blood-2012-01-404004

Published: 2012

Includes: Supplemental data

Journal Articles

STAT3 Is Required for Notch-Induced Leukemogenesis, and Functions As a Critical Survival Effector in T-Cell Leukemia

Jixin Ding, PhD, Ana L. Batista, PhD, Sonia Rodriguez-Rodriguez, PhD, Lin Wang, PhD, Wenjun Zhang, PhD, Akira Moh, PhD, Xin-Yuan Fu, PhD, Weinian Shou, PhD, Nadia Carlesso, MD, PhD, Angelo A Cardoso, MD, PhD

Blood (2011) 118 (21): 920.

https://doi.org/10.1182/blood.V118.21.920.920

Published: 2011

Journal Articles

STAT3 Deletion In Hematopoietic Tissue Causes Mitochondrial Dysfunction with Increased ROS Leading to LT-Repopulating Stem Cell Depletion, Erythroid Dysplasia, Peripheral Neutrophilia, and a Rapid Aging-Like Phenotype.

Charlie Mantel, Steve Messina-Graham, Akira Moh, PhD, Xin-Yuan Fu, PhD, Hal E. Broxmeyer, PhD

Blood (2010) 116 (21): 2621.

https://doi.org/10.1182/blood.V116.21.2621.2621

Published: 2010

Journal Articles

The Earliest Stages of Loss of Stem Cell Self-Renewal in-Vivo Is Linked to Upregulated Biosynthesis of “Quiet” Mitochondria and Is Influenced by CXCR4 Activation and STAT3 Gene Deletion.

Charlie Mantel, Steven Messina-Graham, Akira Moh, PhD, Xin-Yuan Fu, PhD, Hal E. Broxmeyer, PhD

Blood (2009) 114 (22): 2546.

https://doi.org/10.1182/blood.V114.22.2546.2546

Published: 2009

Journal Articles

Survivin mediates aberrant hematopoietic progenitor cell proliferation and acute leukemia in mice induced by internal tandem duplication of Flt3

Seiji Fukuda, Pratibha Singh, Akira Moh, Mariko Abe, Edward M. Conway, H. Scott Boswell, Seiji Yamaguchi, Xin-Yuan Fu, Louis M. Pelus

Blood (2009) 114 (2): 394–403.

https://doi.org/10.1182/blood-2008-11-188714

Published: 2009

Includes: Supplemental data

Journal Articles

STAT3 Deletion after Birth Results in Marked Decreases in Absolute Numbers and Cell Cycling Status of Marrow and Spleen Myeloid Progenitor Cells, and STAT3 Is a Critical Molecule in Mediating Synergistically Stimulated Progenitor Cell Proliferation.

Hal E. Broxmeyer, PhD, Scott Cooper, BSc, Giao Hangoc, DVM, Wenjun Zhang, PhD, Akira Moh, PhD, Thomas Welte, PhD, Xin-Yuan Fu, PhD

Blood (2005) 106 (11): 1346.

https://doi.org/10.1182/blood.V106.11.1346.1346

Published: 2005

NARROW

Mouse hematopoietic cell–targeted STAT3 deletion: stem/progenitor cell defects, mitochondrial dysfunction, ROS overproduction, and a rapid aging–like phenotype

STAT3 Is Required for Notch-Induced Leukemogenesis, and Functions As a Critical Survival Effector in T-Cell Leukemia

STAT3 Deletion In Hematopoietic Tissue Causes Mitochondrial Dysfunction with Increased ROS Leading to LT-Repopulating Stem Cell Depletion, Erythroid Dysplasia, Peripheral Neutrophilia, and a Rapid Aging-Like Phenotype.

The Earliest Stages of Loss of Stem Cell Self-Renewal in-Vivo Is Linked to Upregulated Biosynthesis of “Quiet” Mitochondria and Is Influenced by CXCR4 Activation and STAT3 Gene Deletion.

Survivin mediates aberrant hematopoietic progenitor cell proliferation and acute leukemia in mice induced by internal tandem duplication of Flt3

STAT3 Deletion after Birth Results in Marked Decreases in Absolute Numbers and Cell Cycling Status of Marrow and Spleen Myeloid Progenitor Cells, and STAT3 Is a Critical Molecule in Mediating Synergistically Stimulated Progenitor Cell Proliferation.

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ASH Publications

American Society of Hematology

Mouse hematopoietic cell–targeted STAT3 deletion: stem/progenitor cell defects, mitochondrial dysfunction, ROS overproduction, and a rapid aging–like phenotype

STAT3 Is Required for Notch-Induced Leukemogenesis, and Functions As a Critical Survival Effector in T-Cell Leukemia

STAT3 Deletion In Hematopoietic Tissue Causes Mitochondrial Dysfunction with Increased ROS Leading to LT-Repopulating Stem Cell Depletion, Erythroid Dysplasia, Peripheral Neutrophilia, and a Rapid Aging-Like Phenotype.

The Earliest Stages of Loss of Stem Cell Self-Renewal in-Vivo Is Linked to Upregulated Biosynthesis of “Quiet” Mitochondria and Is Influenced by CXCR4 Activation and STAT3 Gene Deletion.

Survivin mediates aberrant hematopoietic progenitor cell proliferation and acute leukemia in mice induced by internal tandem duplication of Flt3

STAT3 Deletion after Birth Results in Marked Decreases in Absolute Numbers and Cell Cycling Status of Marrow and Spleen Myeloid Progenitor Cells, and STAT3 Is a Critical Molecule in Mediating Synergistically Stimulated Progenitor Cell Proliferation.

Email alerts

ASH Publications

American Society of Hematology

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