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Journal Articles

Hypoxia Inducible Factor-1α Is over Expressed in CLL B Cells Because of an Impaired Proteasome Pathway Associated with Defective Interaction with von Hippel-Landau Protein.

Yean K. Lee, Ann K. Strege, Nancy D. Bone, Linda E. Wellik, D. A. Chan, A. J. Giaccia, Debabrata Mukhopadhyay, MD, PhD, Neil E. Kay, MD

Blood (2005) 106 (11): 2115.

https://doi.org/10.1182/blood.V106.11.2115.2115

Published: 2005

Journal Articles

Adaphostin-induced apoptosis in CLL B cells is associated with induction of oxidative stress and exhibits synergy with fludarabine

Tait D. Shanafelt, Yean K. Lee, Nancy D. Bone, Ann K. Strege, Ven L. Narayanan, Edward A. Sausville, Susan M. Geyer, Scott H. Kaufmann, Neil E. Kay

Blood (2005) 105 (5): 2099–2106.

https://doi.org/10.1182/blood-2004-06-2205

Published: 2005

Journal Articles

Adaphostin-Induced Apoptosis in CLL B-Cells Is Associated with Induction of Oxidative Stress and Exhibits Synergy with Fludarabine.

Tait D. Shanafelt, M.D., Y. Kit Lee, Nancy D. Bone, Ann K. Strege, Scott H. Kaufmann, M.D., Ph.D., Neil E. Kay, M.D.

Blood (2004) 104 (11): 3482.

https://doi.org/10.1182/blood.V104.11.3482.3482

Published: 2004

Journal Articles

CLL B Cell Interaction with Bone Biopsy Generated Marrow Stromal Elements Enhances Their Apoptosis Resistance in Association with an Angiogenic Switch.

Neil E. Kay, MD, Ann K. Strege, BS, Tait D. Shanafelt, MD, Nancy D. Bone, BS, Yean K. Lee, BS

Blood (2004) 104 (11): 1914.

https://doi.org/10.1182/blood.V104.11.1914.1914

Published: 2004

Journal Articles

VEGF receptor phosphorylation status and apoptosis is modulated by a green tea component, epigallocatechin-3-gallate (EGCG), in B-cell chronic lymphocytic leukemia

Yean K. Lee, Nancy D. Bone, Ann K. Strege, Tait D. Shanafelt, Diane F. Jelinek, Neil E. Kay

Blood (2004) 104 (3): 788–794.

https://doi.org/10.1182/blood-2003-08-2763

Published: 2004

NARROW

Hypoxia Inducible Factor-1α Is over Expressed in CLL B Cells Because of an Impaired Proteasome Pathway Associated with Defective Interaction with von Hippel-Landau Protein.

Adaphostin-induced apoptosis in CLL B cells is associated with induction of oxidative stress and exhibits synergy with fludarabine

Adaphostin-Induced Apoptosis in CLL B-Cells Is Associated with Induction of Oxidative Stress and Exhibits Synergy with Fludarabine.

CLL B Cell Interaction with Bone Biopsy Generated Marrow Stromal Elements Enhances Their Apoptosis Resistance in Association with an Angiogenic Switch.

VEGF receptor phosphorylation status and apoptosis is modulated by a green tea component, epigallocatechin-3-gallate (EGCG), in B-cell chronic lymphocytic leukemia

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ASH Publications

American Society of Hematology

Hypoxia Inducible Factor-1α Is over Expressed in CLL B Cells Because of an Impaired Proteasome Pathway Associated with Defective Interaction with von Hippel-Landau Protein.

Adaphostin-induced apoptosis in CLL B cells is associated with induction of oxidative stress and exhibits synergy with fludarabine

Adaphostin-Induced Apoptosis in CLL B-Cells Is Associated with Induction of Oxidative Stress and Exhibits Synergy with Fludarabine.

CLL B Cell Interaction with Bone Biopsy Generated Marrow Stromal Elements Enhances Their Apoptosis Resistance in Association with an Angiogenic Switch.

VEGF receptor phosphorylation status and apoptosis is modulated by a green tea component, epigallocatechin-3-gallate (EGCG), in B-cell chronic lymphocytic leukemia

Email alerts

ASH Publications

American Society of Hematology

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