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Journal Articles

Murine models of erythroid 5ALA synthesis disorders and their conditional synthetic lethal dependency on pyridoxine

Sarah Ducamp, Anoop K. Sendamarai, Dean R. Campagna, Desmond Wai Loon Chin, Yuko Fujiwara, Paul J. Schmidt, Mark D. Fleming

Blood (2024) 144 (13): 1418–1432.

https://doi.org/10.1182/blood.2023023078

Published: 2024

Includes: Supplemental data

Journal Articles

Lack of Gdf11 does not improve anemia or prevent the activity of RAP-536 in a mouse model of β-thalassemia

Amaliris Guerra, Paraskevi Rea Oikonomidou, Sayantani Sinha, Jianbing Zhang, Vania Lo Presti, Callum R. Hamilton, Laura Breda, Carla Casu, Ping La, Ana C. Martins, Anoop K. Sendamarai, Mark Fleming, Stefano Rivella

Blood (2019) 134 (6): 568–572.

https://doi.org/10.1182/blood.2019001057

Published: 2019

Includes: Supplemental data

Journal Articles

A recurring mutation in the respiratory complex 1 protein NDUFB11 is responsible for a novel form of X-linked sideroblastic anemia

Daniel A. Lichtenstein, Andrew W. Crispin, Anoop K. Sendamarai, Dean R. Campagna, Klaus Schmitz-Abe, Cristovao M. Sousa, Martin D. Kafina, Paul J. Schmidt, Charlotte M. Niemeyer, John Porter, Alison May, Mrinal M. Patnaik, Matthew M. Heeney, Alec Kimmelman, Sylvia S. Bottomley, Barry H. Paw, Kyriacos Markianos, Mark D. Fleming

Blood (2016) 128 (15): 1913–1917.

https://doi.org/10.1182/blood-2016-05-719062

Published: 2016

Includes: Supplemental data

Journal Articles

Congenital sideroblastic anemia due to mutations in the mitochondrial HSP70 homologue HSPA9

Blood (2015) 126 (25): 2734–2738.

https://doi.org/10.1182/blood-2015-09-659854

Published: 2015

Includes: Supplemental data

Journal Articles

Mutations in TRNT1 cause congenital sideroblastic anemia with immunodeficiency, fevers, and developmental delay (SIFD)

Blood (2014) 124 (18): 2867–2871.

https://doi.org/10.1182/blood-2014-08-591370

Published: 2014

Includes: Supplemental data

Journal Articles

An RNAi therapeutic targeting Tmprss6 decreases iron overload in Hfe −/− mice and ameliorates anemia and iron overload in murine β-thalassemia intermedia

Paul J. Schmidt, Iva Toudjarska, Anoop K. Sendamarai, Tim Racie, Stuart Milstein, Brian R. Bettencourt, Julia Hettinger, David Bumcrot, Mark D. Fleming

Blood (2013) 121 (7): 1200–1208.

https://doi.org/10.1182/blood-2012-09-453977

Published: 2013

Includes: Supplemental data

Journal Articles

RNAi-Mediated Inhibition of Tmprss6 Ameliorates Anemia and Secondary Iron Overload in a Mouse Model of β-Thalassemia Intermedia and Decreases Iron Overload in Hfe −/− Mice

Paul J Schmidt, PhD, Anoop K Sendamarai, PhD, Ivanka Toudjarska, PhD, Tim Racie, Jim S Butler, PhD, Mark D. Fleming, MD, DPhil, David Bumcrot, PhD

Blood (2012) 120 (21): 1018.

https://doi.org/10.1182/blood.V120.21.1018.1018

Published: 2012

NARROW

Murine models of erythroid 5ALA synthesis disorders and their conditional synthetic lethal dependency on pyridoxine

Lack of Gdf11 does not improve anemia or prevent the activity of RAP-536 in a mouse model of β-thalassemia

A recurring mutation in the respiratory complex 1 protein NDUFB11 is responsible for a novel form of X-linked sideroblastic anemia

Congenital sideroblastic anemia due to mutations in the mitochondrial HSP70 homologue HSPA9

Mutations in TRNT1 cause congenital sideroblastic anemia with immunodeficiency, fevers, and developmental delay (SIFD)

An RNAi therapeutic targeting Tmprss6 decreases iron overload in Hfe −/− mice and ameliorates anemia and iron overload in murine β-thalassemia intermedia

RNAi-Mediated Inhibition of Tmprss6 Ameliorates Anemia and Secondary Iron Overload in a Mouse Model of β-Thalassemia Intermedia and Decreases Iron Overload in Hfe −/− Mice

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ASH Publications

American Society of Hematology

Murine models of erythroid 5ALA synthesis disorders and their conditional synthetic lethal dependency on pyridoxine

Lack of Gdf11 does not improve anemia or prevent the activity of RAP-536 in a mouse model of β-thalassemia

A recurring mutation in the respiratory complex 1 protein NDUFB11 is responsible for a novel form of X-linked sideroblastic anemia

Congenital sideroblastic anemia due to mutations in the mitochondrial HSP70 homologue HSPA9

Mutations in TRNT1 cause congenital sideroblastic anemia with immunodeficiency, fevers, and developmental delay (SIFD)

An RNAi therapeutic targeting Tmprss6 decreases iron overload in Hfe −/− mice and ameliorates anemia and iron overload in murine β-thalassemia intermedia

RNAi-Mediated Inhibition of Tmprss6 Ameliorates Anemia and Secondary Iron Overload in a Mouse Model of β-Thalassemia Intermedia and Decreases Iron Overload in Hfe −/− Mice

Email alerts

ASH Publications

American Society of Hematology

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