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Journal Articles

SOCS1 Cooperates with FLT3-ITD In the Development of Myeloproliferative Disease by Promoting the Escape From External Cytokine Control.

Christian H. Brandts, Pavankumar Reddy, Bülent Sargin, Chunaram Choudhary, Carsten Muller-Tidow, Wolfgang E. Berdel, Hubert Serve

Blood (2010) 116 (21): 1054.

https://doi.org/10.1182/blood.V116.21.1054.1054

Published: 2010

Journal Articles

Kinase Activity of RTKs Is Dispensable for Factor Independent Growth and Transformation of a Myeloid Cell Line 32D in the Presence of Cbl Mutants.

Bülent Sargin on behalf of the Study Alliance Leukemias (SAL), Srinivasa Rao Bandi, Marion Rensinghoff, Rebekka Grundler, Lara Tickenbrock, Justus Duyster, Wolfgang E Berdel, Christian Brandts, MD, Hubert Serve, Carsten Müller-Tidow

Blood (2009) 114 (22): 3970.

https://doi.org/10.1182/blood.V114.22.3970.3970

Published: 2009

Journal Articles

Myeloproliferative Disease or Myeloid Leukemia Caused by Cbl Mutants in a Mouse Transplantation Model.

Bülent Sargin on behalf of the Study Alliance Leukemias (SAL), Srinivasa Rao Bandi, Marion Rensinghoff, Rebekka Grundler, Lara Tickenbrock, Gabriele Köhler, Justus Duyster, Wolfgang E Berdel, Hubert Serve, Christian Brandts, MD, Carsten Müller-Tidow

Blood (2009) 114 (22): 3973.

https://doi.org/10.1182/blood.V114.22.3973.3973

Published: 2009

Journal Articles

Mesenchymal Stems Cells and Regulatory T Cells Inhibit Alloantigen Mediated Graft-Versus-Host Reaction but Also Impair Graft-Versus-Tumor Reactions Targeting Tumor Associated Antigens

Matthias Stelljes, Sarah Harmata, Verena Schlösser, Corinna Opitz, Jörn Albring, Felix Rosenow, Bülent Sargin, Wolfgang E. Berdel, Joachim Kienast

Blood (2008) 112 (11): 4613.

https://doi.org/10.1182/blood.V112.11.4613.4613

Published: 2008

Journal Articles

Cbl Mutants Interact with c-Kit and Lead to Factor Independent Growth and Transformation of a Myeloid Cell Line 32D.

Bülent Sargin, MD, Srinivasa Rao Bandi, Benjamin August, Marion Rensinghoff, Chunaram Choudhary, Pavan Nagaruri Gonchi, Wolfgang E. Berdel, Carsten Müller-Tidow, Hubert Serve

Blood (2007) 110 (11): 2198.

https://doi.org/10.1182/blood.V110.11.2198.2198

Published: 2007

Journal Articles

AML-associated Flt3 kinase domain mutations show signal transduction differences compared with Flt3 ITD mutations

Chunaram Choudhary, Joachim Schwäble, Christian Brandts, Lara Tickenbrock, Bülent Sargin, Thomas Kindler, Thomas Fischer, Wolfgang E. Berdel, Carsten Müller-Tidow, Hubert Serve

Blood (2005) 106 (1): 265–273.

https://doi.org/10.1182/blood-2004-07-2942

Published: 2005

Journal Articles

Flt3 tandem duplication mutations cooperate with Wnt signaling in leukemic signal transduction

Lara Tickenbrock, Joachim Schwäble, Markus Wiedehage, Björn Steffen, Bülent Sargin, Chunaram Choudhary, Christian Brandts, Wolfgang E. Berdel, Carsten Müller-Tidow, Hubert Serve

Blood (2005) 105 (9): 3699–3706.

https://doi.org/10.1182/blood-2004-07-2924

Published: 2005

Journal Articles

RGS2 is an important target gene of Flt3-ITD mutations in AML and functions in myeloid differentiation and leukemic transformation

Joachim Schwäble, Chunaram Choudhary, Christian Thiede, Lara Tickenbrock, Bülent Sargin, Claudia Steur, Maike Rehage, Annika Rudat, Christian Brandts, Wolfgang E. Berdel, Carsten Müller-Tidow, Hubert Serve

Blood (2005) 105 (5): 2107–2114.

https://doi.org/10.1182/blood-2004-03-0940

Published: 2005

Journal Articles

Suppression of myeloid transcription factors and induction of STAT response genes by AML-specific Flt3 mutations

Masao Mizuki, Joachim Schwäble, Claudia Steur, Chunaram Choudhary, Shuchi Agrawal, Bülent Sargin, Björn Steffen, Itaru Matsumura, Yuzuru Kanakura, Frank D. Böhmer, Carsten Müller-Tidow, Wolfgang E. Berdel, Hubert Serve

Blood (2003) 101 (8): 3164–3173.

https://doi.org/10.1182/blood-2002-06-1677

Published: 2003

NARROW

SOCS1 Cooperates with FLT3-ITD In the Development of Myeloproliferative Disease by Promoting the Escape From External Cytokine Control.

Kinase Activity of RTKs Is Dispensable for Factor Independent Growth and Transformation of a Myeloid Cell Line 32D in the Presence of Cbl Mutants.

Myeloproliferative Disease or Myeloid Leukemia Caused by Cbl Mutants in a Mouse Transplantation Model.

Mesenchymal Stems Cells and Regulatory T Cells Inhibit Alloantigen Mediated Graft-Versus-Host Reaction but Also Impair Graft-Versus-Tumor Reactions Targeting Tumor Associated Antigens

Cbl Mutants Interact with c-Kit and Lead to Factor Independent Growth and Transformation of a Myeloid Cell Line 32D.

AML-associated Flt3 kinase domain mutations show signal transduction differences compared with Flt3 ITD mutations

Flt3 tandem duplication mutations cooperate with Wnt signaling in leukemic signal transduction

RGS2 is an important target gene of Flt3-ITD mutations in AML and functions in myeloid differentiation and leukemic transformation

Suppression of myeloid transcription factors and induction of STAT response genes by AML-specific Flt3 mutations

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American Society of Hematology

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ASH Publications

American Society of Hematology

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