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Journal Articles

β 2 integrin–derived signals induce cell survival and proliferation of AML blasts by activating a Syk/STAT signaling axis

Thomas Oellerich, Mark F. Oellerich, Michael Engelke, Silvia Münch, Sebastian Mohr, Marika Nimz, He-Hsuan Hsiao, Jasmin Corso, Jing Zhang, Hanibal Bohnenberger, Tobias Berg, Michael A. Rieger, Jürgen Wienands, Gesine Bug, Christian Brandts, Henning Urlaub, Hubert Serve

Blood (2013) 121 (19): 3889–3899.

https://doi.org/10.1182/blood-2012-09-457887

Published: 2013

Includes: Supplemental data

Journal Articles

AML1/ETO induces self-renewal in hematopoietic progenitor cells via the Groucho-related amino-terminal AES protein

Björn Steffen, Markus Knop, Ulla Bergholz, Olesya Vakhrusheva, Miriam Rode, Gabriele Köhler, Marcel-Philipp Henrichs, Etmar Bulk, Sina Hehn, Martin Stehling, Martin Dugas, Nicole Bäumer, Petra Tschanter, Christian Brandts, Steffen Koschmieder, Wolfgang E. Berdel, Hubert Serve, Carol Stocking, Carsten Müller-Tidow

Blood (2011) 117 (16): 4328–4337.

https://doi.org/10.1182/blood-2009-09-242545

Published: 2011

Includes: Supplemental data

Journal Articles

Sorafenib In Combination with Standard Induction and Consolidation Therapy In Elderly AML Patients: Results From a Randomized, Placebo-Controlled Phase II Trial

Blood (2010) 116 (21): 333.

https://doi.org/10.1182/blood.V116.21.333.333

Published: 2010

Journal Articles

Addition of 5-Azacitidine to Standard Induction Therapy Is Feasible In Older Patients with Acute Myeloid Leukemia: The AML-AZA Trial of the SAL Study Group

Utz O Krug, MD, Anja Koschmieder, Joachim Gerss, PhD, Hubert Serve, MD, Björn Steffen, MD, Gesine Bug, MD, Markus Schaich, MD, Christian Brandts, MD, Christoph Röllig, MD, Gerhard Ehninger, MD, Ulrich Duehrsen, MD, Richard Noppeney, MD, Wolfgang E. Berdel, MD, Steffen Koschmieder, MD, Carsten Müller-Tidow, MD

Blood (2010) 116 (21): 2180.

https://doi.org/10.1182/blood.V116.21.2180.2180

Published: 2010

Journal Articles

Kinase Activity of RTKs Is Dispensable for Factor Independent Growth and Transformation of a Myeloid Cell Line 32D in the Presence of Cbl Mutants.

Bülent Sargin on behalf of the Study Alliance Leukemias (SAL), Srinivasa Rao Bandi, Marion Rensinghoff, Rebekka Grundler, Lara Tickenbrock, Justus Duyster, Wolfgang E Berdel, Christian Brandts, MD, Hubert Serve, Carsten Müller-Tidow

Blood (2009) 114 (22): 3970.

https://doi.org/10.1182/blood.V114.22.3970.3970

Published: 2009

Journal Articles

Myeloproliferative Disease or Myeloid Leukemia Caused by Cbl Mutants in a Mouse Transplantation Model.

Bülent Sargin on behalf of the Study Alliance Leukemias (SAL), Srinivasa Rao Bandi, Marion Rensinghoff, Rebekka Grundler, Lara Tickenbrock, Gabriele Köhler, Justus Duyster, Wolfgang E Berdel, Hubert Serve, Christian Brandts, MD, Carsten Müller-Tidow

Blood (2009) 114 (22): 3973.

https://doi.org/10.1182/blood.V114.22.3973.3973

Published: 2009

Journal Articles

E3 ligase–defective Cbl mutants lead to a generalized mastocytosis and myeloproliferative disease

Srinivasa Rao Bandi, Christian Brandts, Marion Rensinghoff, Rebekka Grundler, Lara Tickenbrock, Gabriele Köhler, Justus Duyster, Wolfgang E. Berdel, Carsten Müller-Tidow, Hubert Serve, on behalf of the Study Alliance Leukemias, Bülent Sargin

Blood (2009) 114 (19): 4197–4208.

https://doi.org/10.1182/blood-2008-12-190934

Published: 2009

Includes: Supplemental data

Journal Articles

Frequent CBL mutations associated with 11q acquired uniparental disomy in myeloproliferative neoplasms

Francis H. Grand, Claire E. Hidalgo-Curtis, Thomas Ernst, Katerina Zoi, Christine Zoi, Carolann McGuire, Sebastian Kreil, Amy Jones, Joannah Score, Georgia Metzgeroth, David Oscier, Andrew Hall, Christian Brandts, Hubert Serve, Andreas Reiter, Andrew J. Chase, Nicholas C. P. Cross

Blood (2009) 113 (24): 6182–6192.

https://doi.org/10.1182/blood-2008-12-194548

Published: 2009

Journal Articles

Activation of AKT1 in Hematopoietic Stem Cells Causes a Myeloproliferative Disease in a Tet-Inducible Mouse Model.

Christian Brandts, MD, Miriam Rode, PhD, Beate Lindtner, MA, Gabriele Koehler, MD, Steffen Koschmieder, MD, Carsten Mueller-Tidow, Wolfgang E. Berdel, Hubert Serve

Blood (2008) 112 (11): 1356.

https://doi.org/10.1182/blood.V112.11.1356.1356

Published: 2008

Journal Articles

Frequent CBL Mutations Associated with 11q Acquired Uniparental Disomy in Myeloproliferative Neoplasms

Francis Grand, PhD, Claire Hidalgo-Curtis, BSc, Thomas Ernst, MD, Katerina Zoi, PhD, Christine Zoi, MD, Carolann McGuire, PhD, Sebastian Kreil, MD, Georgia Metzgeroth, MD, David Oscier, MD, Andrew Hall, PhD, Christian Brandts, MD, Hubert Serve, Andreas Reiter, MD, Andrew Chase, PhD, Nicholas C.P. Cross, PhD

Blood (2008) 112 (11): 174.

https://doi.org/10.1182/blood.V112.11.174.174

Published: 2008

Journal Articles

Flt3-dependent transformation by inactivating c-Cbl mutations in AML

Bülent Sargin, Chunaram Choudhary, Nicola Crosetto, Mirko H. H. Schmidt, Rebekka Grundler, Marion Rensinghoff, Christine Thiessen, Lara Tickenbrock, Joachim Schwäble, Christian Brandts, Benjamin August, Steffen Koschmieder, Srinivasa Rao Bandi, Justus Duyster, Wolfgang E. Berdel, Carsten Müller-Tidow, Ivan Dikic, Hubert Serve

Blood (2007) 110 (3): 1004–1012.

https://doi.org/10.1182/blood-2007-01-066076

Published: 2007

Journal Articles

Activation mechanisms of STAT5 by oncogenic Flt3-ITD

Chunaram Choudhary, Christian Brandts, Joachim Schwable, Lara Tickenbrock, Bülent Sargin, Andrea Ueker, Frank-D. Böhmer, Wolfgang E. Berdel, Carsten Müller-Tidow, Hubert Serve

Blood (2007) 110 (1): 370–374.

https://doi.org/10.1182/blood-2006-05-024018

Published: 2007

Includes: Supplemental data

Journal Articles

Activation Mechanisms of STAT5 by Oncogenic Flt3-ITD.

Chunaram Choudhary, Christian Brandts, Joachim Schwable, Lara Tickenbrock, Bulent Sargin, Andrea Ueker, Frank D. Bohmer, Wolfgang E. Berdel, Carsten Müller-Tidow, Hubert Serve

Blood (2006) 108 (11): 1435.

https://doi.org/10.1182/blood.V108.11.1435.1435

Published: 2006

Journal Articles

Clinical resistance to the kinase inhibitor PKC412 in acute myeloid leukemia by mutation of Asn-676 in the FLT3 tyrosine kinase domain

Blood (2006) 107 (1): 293–300.

https://doi.org/10.1182/blood-2005-06-2469

Published: 2006

Journal Articles

AML-associated Flt3 kinase domain mutations show signal transduction differences compared with Flt3 ITD mutations

Chunaram Choudhary, Joachim Schwäble, Christian Brandts, Lara Tickenbrock, Bülent Sargin, Thomas Kindler, Thomas Fischer, Wolfgang E. Berdel, Carsten Müller-Tidow, Hubert Serve

Blood (2005) 106 (1): 265–273.

https://doi.org/10.1182/blood-2004-07-2942

Published: 2005

Journal Articles

Flt3 tandem duplication mutations cooperate with Wnt signaling in leukemic signal transduction

Lara Tickenbrock, Joachim Schwäble, Markus Wiedehage, Björn Steffen, Bülent Sargin, Chunaram Choudhary, Christian Brandts, Wolfgang E. Berdel, Carsten Müller-Tidow, Hubert Serve

Blood (2005) 105 (9): 3699–3706.

https://doi.org/10.1182/blood-2004-07-2924

Published: 2005

Journal Articles

RGS2 is an important target gene of Flt3-ITD mutations in AML and functions in myeloid differentiation and leukemic transformation

Joachim Schwäble, Chunaram Choudhary, Christian Thiede, Lara Tickenbrock, Bülent Sargin, Claudia Steur, Maike Rehage, Annika Rudat, Christian Brandts, Wolfgang E. Berdel, Carsten Müller-Tidow, Hubert Serve

Blood (2005) 105 (5): 2107–2114.

https://doi.org/10.1182/blood-2004-03-0940

Published: 2005

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β 2 integrin–derived signals induce cell survival and proliferation of AML blasts by activating a Syk/STAT signaling axis

AML1/ETO induces self-renewal in hematopoietic progenitor cells via the Groucho-related amino-terminal AES protein

Sorafenib In Combination with Standard Induction and Consolidation Therapy In Elderly AML Patients: Results From a Randomized, Placebo-Controlled Phase II Trial

Addition of 5-Azacitidine to Standard Induction Therapy Is Feasible In Older Patients with Acute Myeloid Leukemia: The AML-AZA Trial of the SAL Study Group

Kinase Activity of RTKs Is Dispensable for Factor Independent Growth and Transformation of a Myeloid Cell Line 32D in the Presence of Cbl Mutants.

Myeloproliferative Disease or Myeloid Leukemia Caused by Cbl Mutants in a Mouse Transplantation Model.

E3 ligase–defective Cbl mutants lead to a generalized mastocytosis and myeloproliferative disease

Frequent CBL mutations associated with 11q acquired uniparental disomy in myeloproliferative neoplasms

Activation of AKT1 in Hematopoietic Stem Cells Causes a Myeloproliferative Disease in a Tet-Inducible Mouse Model.

Frequent CBL Mutations Associated with 11q Acquired Uniparental Disomy in Myeloproliferative Neoplasms

Flt3-dependent transformation by inactivating c-Cbl mutations in AML

Activation mechanisms of STAT5 by oncogenic Flt3-ITD

Activation Mechanisms of STAT5 by Oncogenic Flt3-ITD.

Clinical resistance to the kinase inhibitor PKC412 in acute myeloid leukemia by mutation of Asn-676 in the FLT3 tyrosine kinase domain

AML-associated Flt3 kinase domain mutations show signal transduction differences compared with Flt3 ITD mutations

Flt3 tandem duplication mutations cooperate with Wnt signaling in leukemic signal transduction

RGS2 is an important target gene of Flt3-ITD mutations in AML and functions in myeloid differentiation and leukemic transformation

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American Society of Hematology

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