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Journal Articles

Thymidylate Synthase (TS) Expression In Diffuse Large B-Cell Lymphoma (DLBCL): Identification of a Potential Therapeutic Target

Emilian Racila, Sergei I Syrbu, MD, Michel Nasr, MD, Malik E. Juweid, MD

Blood (2010) 116 (21): 5107.

https://doi.org/10.1182/blood.V116.21.5107.5107

Published: 2010

Journal Articles

Depletion of the C3 component of complement enhances the ability of rituximab-coated target cells to activate human NK cells and improves the efficacy of monoclonal antibody therapy in an in vivo model

Siao-Yi Wang, Suresh Veeramani, Emilian Racila, Jeffrey Cagley, David C. Fritzinger, Carl-Wilhelm Vogel, William St John, George J. Weiner

Blood (2009) 114 (26): 5322–5330.

https://doi.org/10.1182/blood-2009-01-200469

Published: 2009

Journal Articles

NK-cell activation and antibody-dependent cellular cytotoxicity induced by rituximab-coated target cells is inhibited by the C3b component of complement

Siao-Yi Wang, Emilian Racila, Ronald P. Taylor, George J. Weiner

Blood (2008) 111 (3): 1456–1463.

https://doi.org/10.1182/blood-2007-02-074716

Published: 2008

Journal Articles

The C3b Component of Complement Inhibits NK Cell Activation Induced by Rituximab-Coated Target Cells.

Siao-Yi Wang, BA, Emilian Racila, MD, Ronald P. Taylor, PhD, George J. Weiner, MD

Blood (2006) 108 (11): 2505.

https://doi.org/10.1182/blood.V108.11.2505.2505

Published: 2006

Journal Articles

A Polymorphism in the C1qA Component of Complement Correlates with Prolonged Complete Remission Following Rituximab Therapy of Follicular Lymphoma.

Emilian Racila, MD, Wen-Kai Weng, MD, James E. Wooldridge, MD, Brian K. Link, MD, Ronald Levy, MD, George J. Weiner, MD

Blood (2005) 106 (11): 778.

https://doi.org/10.1182/blood.V106.11.778.778

Published: 2005

Journal Articles

Tumor Dormancy and Cell Signaling. V. Regrowth of the BCL 1 Tumor After Dormancy Is Established

Ellen S. Vitetta, Thomas F. Tucker, Emilian Racila, Yi-Wu Huang, Radu Marches, Nancy Lane, Richard H. Scheuermann, Nancy E. Street, Takeshi Watanabe, Jonathan W. Uhr

Blood (1997) 89 (12): 4425–4436.

https://doi.org/10.1182/blood.V89.12.4425

Published: 1997

NARROW

Thymidylate Synthase (TS) Expression In Diffuse Large B-Cell Lymphoma (DLBCL): Identification of a Potential Therapeutic Target

Depletion of the C3 component of complement enhances the ability of rituximab-coated target cells to activate human NK cells and improves the efficacy of monoclonal antibody therapy in an in vivo model

NK-cell activation and antibody-dependent cellular cytotoxicity induced by rituximab-coated target cells is inhibited by the C3b component of complement

The C3b Component of Complement Inhibits NK Cell Activation Induced by Rituximab-Coated Target Cells.

A Polymorphism in the C1qA Component of Complement Correlates with Prolonged Complete Remission Following Rituximab Therapy of Follicular Lymphoma.

Tumor Dormancy and Cell Signaling. V. Regrowth of the BCL 1 Tumor After Dormancy Is Established

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ASH Publications

American Society of Hematology

Thymidylate Synthase (TS) Expression In Diffuse Large B-Cell Lymphoma (DLBCL): Identification of a Potential Therapeutic Target

Depletion of the C3 component of complement enhances the ability of rituximab-coated target cells to activate human NK cells and improves the efficacy of monoclonal antibody therapy in an in vivo model

NK-cell activation and antibody-dependent cellular cytotoxicity induced by rituximab-coated target cells is inhibited by the C3b component of complement

The C3b Component of Complement Inhibits NK Cell Activation Induced by Rituximab-Coated Target Cells.

A Polymorphism in the C1qA Component of Complement Correlates with Prolonged Complete Remission Following Rituximab Therapy of Follicular Lymphoma.

Tumor Dormancy and Cell Signaling. V. Regrowth of the BCL 1 Tumor After Dormancy Is Established

Email alerts

ASH Publications

American Society of Hematology

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