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Journal Articles

BCL6 Is Required for Leukemia-Initiation and Self-Renewal Signaling in Chronic Myeloid Leukemia.

Christian Hurtz, Cihangir Duy, Leandro Cerchietti, Eugene Park, Weimin Ci, PhD, Srividya Swaminathan, Soo-mi Kweon, Lars Klemm, Yong-mi Kim, Giovanni Martinelli, Wolf-Karsten Hofmann, B. Hilda Ye, PhD, Ari Melnick, Markus Müschen

Blood (2009) 114 (22): 2167.

https://doi.org/10.1182/blood.V114.22.2167.2167

Published: 2009

Journal Articles

BCL6-Dependent Negative Regulation of Cell Cycle Checkpoint Regulators Enables Drug-Resistance in Ph + Acute Lymphoblastic Leukemia.

Cihangir Duy, Leandro Cerchietti, J. Jessica Yu, Weimin Ci, PhD, Srividya Swaminathan, Rahul R. Nahar, Soo-mi Kweon, Lars Klemm, B. Hilda Ye, PhD, Ari Melnick, Markus Müschen

Blood (2009) 114 (22): 765.

https://doi.org/10.1182/blood.V114.22.765.765

Published: 2009

Journal Articles

The BCL6 transcriptional program features repression of multiple oncogenes in primary B cells and is deregulated in DLBCL

Weimin Ci, Jose M. Polo, Leandro Cerchietti, Rita Shaknovich, Ling Wang, Shao Ning Yang, Kenny Ye, Pedro Farinha, Douglas E. Horsman, Randy D. Gascoyne, Olivier Elemento, Ari Melnick

Blood (2009) 113 (22): 5536–5548.

https://doi.org/10.1182/blood-2008-12-193037

Published: 2009

Includes: Supplemental data

Journal Articles

Reversible disruption of BCL6 repression complexes by CD40 signaling in normal and malignant B cells

Jose M. Polo, Weimin Ci, Jonathan D. Licht, Ari Melnick

Blood (2008) 112 (3): 644–651.

https://doi.org/10.1182/blood-2008-01-131813

Published: 2008

Includes: Supplemental data

Journal Articles

A “Survival of the Fittest” Mechanism for Weeding Out Potentially Lymphomagenic B-Cells during Germinal Center B-Cell Differentiation.

Weimin Ci, Jose M. Polo, Stella M. Ranuncolo, Ari Melnick

Blood (2007) 110 (11): 559.

https://doi.org/10.1182/blood.V110.11.559.559

Published: 2007

NARROW

BCL6 Is Required for Leukemia-Initiation and Self-Renewal Signaling in Chronic Myeloid Leukemia.

BCL6-Dependent Negative Regulation of Cell Cycle Checkpoint Regulators Enables Drug-Resistance in Ph + Acute Lymphoblastic Leukemia.

The BCL6 transcriptional program features repression of multiple oncogenes in primary B cells and is deregulated in DLBCL

Reversible disruption of BCL6 repression complexes by CD40 signaling in normal and malignant B cells

A “Survival of the Fittest” Mechanism for Weeding Out Potentially Lymphomagenic B-Cells during Germinal Center B-Cell Differentiation.

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ASH Publications

American Society of Hematology

BCL6 Is Required for Leukemia-Initiation and Self-Renewal Signaling in Chronic Myeloid Leukemia.

BCL6-Dependent Negative Regulation of Cell Cycle Checkpoint Regulators Enables Drug-Resistance in Ph + Acute Lymphoblastic Leukemia.

The BCL6 transcriptional program features repression of multiple oncogenes in primary B cells and is deregulated in DLBCL

Reversible disruption of BCL6 repression complexes by CD40 signaling in normal and malignant B cells

A “Survival of the Fittest” Mechanism for Weeding Out Potentially Lymphomagenic B-Cells during Germinal Center B-Cell Differentiation.

Email alerts

ASH Publications

American Society of Hematology

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