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Journal Articles

RUNX1/AML1 DNA-binding domain and ETO/MTG8 NHR2-dimerization domain are critical to AML1-ETO9a leukemogenesis

Ming Yan, Eun-Young Ahn, Scott W. Hiebert, Dong-Er Zhang

Blood (2009) 113 (4): 883–886.

https://doi.org/10.1182/blood-2008-04-153742

Published: 2009

Includes: Supplemental data

Journal Articles

Mutational Analysis of the CBFβ-SMMHC Assembly Competence Domain Identifies a Surface Critical for Multimerization and Inhibition of RUNX1/AML1.

Linsheng Zhang, Jenice D’Costa, Tanawan Kummalue, Isabel Moreno, Curt I. Civin, Scott W. Hiebert, Alan D. Friedman

Blood (2005) 106 (11): 2853.

https://doi.org/10.1182/blood.V106.11.2853.2853

Published: 2005

Journal Articles

RUNX1 Directly Regulates Band 3 Transcription.

Jingping Xie, PhD, Scott W. Hiebert, PhD, Mark J. Koury, MD, Stephen J. Brandt, MD

Blood (2005) 106 (11): 1735.

https://doi.org/10.1182/blood.V106.11.1735.1735

Published: 2005

Journal Articles

Proteomic Identification of TAL1/SCL-Interacting Proteins: ETO-2 and MTGR1 Interact with TAL1 in Erythroid Progenitors.

Ying Cai, Ph.D., Zhixiong Xu, Ph.D., Jingping Xie, Ph.D., Mark J. Koury, M.D., Scott W. Hiebert, Ph.D., Stephen J. Brandt, M.D.

Blood (2004) 104 (11): 357.

https://doi.org/10.1182/blood.V104.11.357.357

Published: 2004

Journal Articles

Neurofibromatosis Type 1 ( NF1 ), a Tumor Suppressor Gene, Is Synergistically Activated by RUNX1, C/EBPαa and ETS2 and Specifically Repressed by the T(8;21) Fusion Protein in Acute Myeloid Leukemia.

Genyan Yang, MD/PhD, Waleed Khalaf, Wade D. Clapp, MD, Scott W. Hiebert, PhD

Blood (2004) 104 (11): 2551.

https://doi.org/10.1182/blood.V104.11.2551.2551

Published: 2004

Journal Articles

Multimerization and Corepression Mediated by the CBFβ-SMMHC Assembly Competence Domain Are Partially Separable and Corepression Is Required to Inhibit Core Binding Factor Activities.

Linsheng Zhang, Ph.D., Scott W. Hiebert, Ph.D., Alan D. Friedman, M.D.

Blood (2004) 104 (11): 1972.

https://doi.org/10.1182/blood.V104.11.1972.1972

Published: 2004

Journal Articles

AML-1/ETO fusion protein is a dominant negative inhibitor of transcriptional repression by the promyelocytic leukemia zinc finger protein

Ari Melnick, Graeme W. Carlile, Melanie J. McConnell, Adam Polinger, Scott W. Hiebert, Jonathan D. Licht

Blood (2000) 96 (12): 3939–3947.

https://doi.org/10.1182/blood.V96.12.3939

Published: 2000

NARROW

RUNX1/AML1 DNA-binding domain and ETO/MTG8 NHR2-dimerization domain are critical to AML1-ETO9a leukemogenesis

Mutational Analysis of the CBFβ-SMMHC Assembly Competence Domain Identifies a Surface Critical for Multimerization and Inhibition of RUNX1/AML1.

RUNX1 Directly Regulates Band 3 Transcription.

Proteomic Identification of TAL1/SCL-Interacting Proteins: ETO-2 and MTGR1 Interact with TAL1 in Erythroid Progenitors.

Neurofibromatosis Type 1 ( NF1 ), a Tumor Suppressor Gene, Is Synergistically Activated by RUNX1, C/EBPαa and ETS2 and Specifically Repressed by the T(8;21) Fusion Protein in Acute Myeloid Leukemia.

Multimerization and Corepression Mediated by the CBFβ-SMMHC Assembly Competence Domain Are Partially Separable and Corepression Is Required to Inhibit Core Binding Factor Activities.

AML-1/ETO fusion protein is a dominant negative inhibitor of transcriptional repression by the promyelocytic leukemia zinc finger protein

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ASH Publications

American Society of Hematology

RUNX1/AML1 DNA-binding domain and ETO/MTG8 NHR2-dimerization domain are critical to AML1-ETO9a leukemogenesis

Mutational Analysis of the CBFβ-SMMHC Assembly Competence Domain Identifies a Surface Critical for Multimerization and Inhibition of RUNX1/AML1.

RUNX1 Directly Regulates Band 3 Transcription.

Proteomic Identification of TAL1/SCL-Interacting Proteins: ETO-2 and MTGR1 Interact with TAL1 in Erythroid Progenitors.

Neurofibromatosis Type 1 ( NF1 ), a Tumor Suppressor Gene, Is Synergistically Activated by RUNX1, C/EBPαa and ETS2 and Specifically Repressed by the T(8;21) Fusion Protein in Acute Myeloid Leukemia.

Multimerization and Corepression Mediated by the CBFβ-SMMHC Assembly Competence Domain Are Partially Separable and Corepression Is Required to Inhibit Core Binding Factor Activities.

AML-1/ETO fusion protein is a dominant negative inhibitor of transcriptional repression by the promyelocytic leukemia zinc finger protein

Email alerts

ASH Publications

American Society of Hematology

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